Ana Persson

473 total citations
7 papers, 305 citations indexed

About

Ana Persson is a scholar working on Cancer Research, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Ana Persson has authored 7 papers receiving a total of 305 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Cancer Research, 3 papers in Molecular Biology and 3 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Ana Persson's work include Protease and Inhibitor Mechanisms (5 papers), Cerebrovascular and Carotid Artery Diseases (3 papers) and Atherosclerosis and Cardiovascular Diseases (3 papers). Ana Persson is often cited by papers focused on Protease and Inhibitor Mechanisms (5 papers), Cerebrovascular and Carotid Artery Diseases (3 papers) and Atherosclerosis and Cardiovascular Diseases (3 papers). Ana Persson collaborates with scholars based in Sweden, United Kingdom and Germany. Ana Persson's co-authors include Mihaela Nitulescu, Isabel Gonçalves, Andreas Edsfeldt, Harry Björkbacka, Marie Nilsson, Helena Grufman, Jan Nilsson, Katarina Berg, Na Young Ko and Jerzy Adamski and has published in prestigious journals such as Nature Communications, PLoS ONE and Stroke.

In The Last Decade

Ana Persson

7 papers receiving 304 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Ana Persson Sweden	6	91	81	74	67	60	7	305
Chihiro Horimai Japan	7	155 1.7×	45 0.6×	47 0.6×	61 0.9×	38 0.6×	9	378
Jun Zou China	13	143 1.6×	42 0.5×	32 0.4×	71 1.1×	71 1.2×	27	400
Niki Prakoura France	13	171 1.9×	97 1.2×	29 0.4×	76 1.1×	62 1.0×	23	436
Tomokazu Ikemoto Japan	11	116 1.3×	47 0.6×	24 0.3×	115 1.7×	56 0.9×	22	321
Houyong Dai China	11	147 1.6×	47 0.6×	26 0.4×	74 1.1×	46 0.8×	20	342
Shengshuai Shan United States	10	72 0.8×	69 0.9×	29 0.4×	35 0.5×	43 0.7×	22	296
Dominika Łacheta Poland	5	66 0.7×	69 0.9×	53 0.7×	33 0.5×	24 0.4×	8	279
Alf‐Åge R. Pettersen Norway	13	112 1.2×	136 1.7×	37 0.5×	233 3.5×	28 0.5×	22	485
Sara Oppi Italy	8	111 1.2×	106 1.3×	59 0.8×	40 0.6×	32 0.5×	15	371
Marco Antonio Peña‐Duque Mexico	12	83 0.9×	80 1.0×	27 0.4×	172 2.6×	27 0.5×	51	389

Countries citing papers authored by Ana Persson

Since Specialization

Citations

This map shows the geographic impact of Ana Persson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ana Persson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ana Persson more than expected).

Fields of papers citing papers by Ana Persson

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ana Persson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ana Persson. The network helps show where Ana Persson may publish in the future.

Co-authorship network of co-authors of Ana Persson

This figure shows the co-authorship network connecting the top 25 collaborators of Ana Persson. A scholar is included among the top collaborators of Ana Persson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ana Persson. Ana Persson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

7 of 7 papers shown

1.

Singh, Pratibha, Jiangming Sun, Michele Cavalera, et al.. (2024). Dysregulation of MMP2-dependent TGF-ß2 activation impairs fibrous cap formation in type 2 diabetes-associated atherosclerosis. Nature Communications. 15(1). 10464–10464. 4 indexed citations

2.

Bengtsson, Eva, Karin Hultman, Andreas Edsfeldt, et al.. (2020). CD163+ macrophages are associated with a vulnerable plaque phenotype in human carotid plaques. Scientific Reports. 10(1). 14362–14362. 28 indexed citations

3.

Hultman, Karin, Andreas Edsfeldt, Harry Björkbacka, et al.. (2019). Cartilage Oligomeric Matrix Protein Associates With a Vulnerable Plaque Phenotype in Human Atherosclerotic Plaques. Stroke. 50(11). 3289–3292. 14 indexed citations

4.

Asciutto, Giuseppe, Nuno Dias, Andreas Edsfeldt, et al.. (2015). Low Elastin Content of Carotid Plaques Is Associated with Increased Risk of Ipsilateral Stroke. PLoS ONE. 10(3). e0121086–e0121086. 16 indexed citations

5.

Edsfeldt, Andreas, Mihaela Nitulescu, Helena Grufman, et al.. (2012). Soluble Urokinase Plasminogen Activator Receptor is Associated With Inflammation in the Vulnerable Human Atherosclerotic Plaque. Stroke. 43(12). 3305–3312. 83 indexed citations

6.

Gonçalves, Isabel, Andreas Edsfeldt, Na Young Ko, et al.. (2012). Evidence Supporting a Key Role of Lp-PLA2-Generated Lysophosphatidylcholine in Human Atherosclerotic Plaque Inflammation. Arteriosclerosis Thrombosis and Vascular Biology. 32(6). 1505–1512. 154 indexed citations

7.

Edsfeldt, Andreas, Nuno Dias, Barbara Elmståhl, et al.. (2011). Low Carotid Calcium Score Is Associated With Higher Levels of Glycosaminoglycans, Tumor Necrosis Factor-Alpha, and Parathyroid Hormone in Human Carotid Plaques. Stroke. 42(10). 2966–2969. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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