Ana Banito

8.5k total citations · 1 hit paper
27 papers, 3.5k citations indexed

About

Ana Banito is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Oncology. According to data from OpenAlex, Ana Banito has authored 27 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 8 papers in Pulmonary and Respiratory Medicine and 8 papers in Oncology. Recurrent topics in Ana Banito's work include Sarcoma Diagnosis and Treatment (8 papers), Cancer-related Molecular Pathways (6 papers) and Telomeres, Telomerase, and Senescence (6 papers). Ana Banito is often cited by papers focused on Sarcoma Diagnosis and Treatment (8 papers), Cancer-related Molecular Pathways (6 papers) and Telomeres, Telomerase, and Senescence (6 papers). Ana Banito collaborates with scholars based in United States, United Kingdom and Germany. Ana Banito's co-authors include Jesús Gil, Juan Carlos Acosta, Selina Raguz, Ana O’Loghlen, Marco Da Costa, David Bernard, Eva Hernando, Marzia Fumagalli, Yoshihiro Takatsu and Fabrizio d’Adda di Fagagna and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

In The Last Decade

Ana Banito

25 papers receiving 3.4k citations

Hit Papers

Chemokine Signaling via the CXCR2 Receptor Reinforces Sen... 2008 2026 2014 2020 2008 400 800 1.2k

Peers

Ana Banito
Juan Carlos Acosta United Kingdom
Ana Banito
Citations per year, relative to Ana Banito Ana Banito (= 1×) peers Juan Carlos Acosta

Countries citing papers authored by Ana Banito

Since Specialization
Citations

This map shows the geographic impact of Ana Banito's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ana Banito with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ana Banito more than expected).

Fields of papers citing papers by Ana Banito

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ana Banito. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ana Banito. The network helps show where Ana Banito may publish in the future.

Co-authorship network of co-authors of Ana Banito

This figure shows the co-authorship network connecting the top 25 collaborators of Ana Banito. A scholar is included among the top collaborators of Ana Banito based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ana Banito. Ana Banito is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Benabdallah, Nezha S., Li Li, Jinxiu Li, et al.. (2025). DNA demethylating agents suppress preclinical models of synovial sarcoma. Journal of Clinical Investigation. 135(13). 1 indexed citations
2.
Will, Rainer, Dominic Helm, Felix Bestvater, et al.. (2025). Loss of SMARCB1 evokes targetable epigenetic vulnerabilities in epithelioid sarcoma. Cancer Communications. 45(5). 494–499.
3.
Benabdallah, Nezha S., R. Wilder Scott, F. Kommoss, et al.. (2023). Aberrant gene activation in synovial sarcoma relies on SSX specificity and increased PRC1.1 stability. Nature Structural & Molecular Biology. 30(11). 1640–1652. 11 indexed citations
4.
Zhu, Changyu, Yadira M. Soto-Feliciano, John P. Morris, et al.. (2023). MLL3 regulates the CDKN2A tumor suppressor locus in liver cancer. eLife. 12. 13 indexed citations
5.
Huang, Chun‐Hao, Francisco J. Sánchez‐Rivera, Margaret C. Kennedy, et al.. (2022). A preclinical platform for assessing antitumor effects and systemic toxicities of cancer drug targets. Proceedings of the National Academy of Sciences. 119(17). e2110557119–e2110557119. 8 indexed citations
6.
Li, Jinxiu, Jared J. Barrott, Mary L. Nelson, et al.. (2021). A Role for SMARCB1 in Synovial Sarcomagenesis Reveals That SS18–SSX Induces Canonical BAF Destruction. Cancer Discovery. 11(10). 2620–2637. 32 indexed citations
7.
Kommoss, F., Kenneth Tou En Chang, Damian Stichel, et al.. (2020). Endometrial stromal sarcomas with BCOR‐rearrangement harbor MDM2 amplifications. The Journal of Pathology Clinical Research. 6(3). 178–184. 36 indexed citations
8.
Loizou, Evangelia, Ana Banito, Geulah Livshits, et al.. (2019). A Gain-of-Function p53-Mutant Oncogene Promotes Cell Fate Plasticity and Myeloid Leukemia through the Pluripotency Factor FOXH1. Cancer Discovery. 9(7). 962–979. 59 indexed citations
9.
Jones, David, Ana Banito, Thomas G. P. Grünewald, et al.. (2019). Molecular characteristics and therapeutic vulnerabilities across paediatric solid tumours. Nature reviews. Cancer. 19(8). 420–438. 85 indexed citations
10.
Banito, Ana, Xiang Li, Jae‐Seok Roe, et al.. (2018). The SS18-SSX Oncoprotein Hijacks KDM2B-PRC1.1 to Drive Synovial Sarcoma. Cancer Cell. 33(3). 527–541.e8. 101 indexed citations
11.
Aarts, Marieke, Athena Georgilis, Meryam Beniazza, et al.. (2017). Coupling shRNA screens with single-cell RNA-seq identifies a dual role for mTOR in reprogramming-induced senescence. Genes & Development. 31(20). 2085–2098. 45 indexed citations
12.
Tasdemir, Nilgun, Ana Banito, Jae‐Seok Roe, et al.. (2016). BRD4 Connects Enhancer Remodeling to Senescence Immune Surveillance. Cancer Discovery. 6(6). 612–629. 278 indexed citations
13.
Tschaharganeh, Darjus F., Wen Xue, Diego F. Calvisi, et al.. (2016). p53-Dependent Nestin Regulation Links Tumor Suppression to Cellular Plasticity in Liver Cancer. Cell. 165(6). 1546–1547. 21 indexed citations
14.
Marqués‐Torrejón, María Ángeles, Eva Porlan, Ana Banito, et al.. (2012). Cyclin-Dependent Kinase Inhibitor p21 Controls Adult Neural Stem Cell Expansion by Regulating Sox2 Gene Expression. Cell stem cell. 12(1). 88–100. 170 indexed citations
15.
Banito, Ana & Jesús Gil. (2010). Induced pluripotent stem cells and senescence: learning the biology to improve the technology. EMBO Reports. 11(5). 353–359. 83 indexed citations
16.
Barradas, Marta, Emma Anderton, Juan Carlos Acosta, et al.. (2009). Histone demethylase JMJD3 contributes to epigenetic control of INK4a/ARF by oncogenic RAS. Genes & Development. 23(10). 1177–1182. 271 indexed citations
17.
Banito, Ana, S. Tamir Rashid, Juan Carlos Acosta, et al.. (2009). Senescence impairs successful reprogramming to pluripotent stem cells. Genes & Development. 23(18). 2134–2139. 489 indexed citations
18.
Cavaco, Branca, Carmo Martins, Ana Banito, et al.. (2008). Familial non-medullary thyroid carcinoma (FNMTC): analysis of fPTC/PRN, NMTC1, MNG1 and TCO susceptibility loci and identification of somatic BRAF and RAS mutations. Endocrine Related Cancer. 15(1). 207–215. 48 indexed citations
19.
Acosta, Juan Carlos, Ana O’Loghlen, Ana Banito, et al.. (2008). Chemokine Signaling via the CXCR2 Receptor Reinforces Senescence. Cell. 133(6). 1006–1018. 1374 indexed citations breakdown →
20.
Banito, Ana, António E. Pinto, Carla Espadinha, Ana Rita Marques, & Valeriano Leite. (2007). Aneuploidy and RAS mutations are mutually exclusive events in the development of well‐differentiated thyroid follicular tumours. Clinical Endocrinology. 67(5). 706–711. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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