Amy DeMicco

404 total citations
12 papers, 293 citations indexed

About

Amy DeMicco is a scholar working on Oncology, Molecular Biology and Cancer Research. According to data from OpenAlex, Amy DeMicco has authored 12 papers receiving a total of 293 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Oncology, 3 papers in Molecular Biology and 3 papers in Cancer Research. Recurrent topics in Amy DeMicco's work include Cancer-related Molecular Pathways (4 papers), Acute Myeloid Leukemia Research (2 papers) and DNA Repair Mechanisms (2 papers). Amy DeMicco is often cited by papers focused on Cancer-related Molecular Pathways (4 papers), Acute Myeloid Leukemia Research (2 papers) and DNA Repair Mechanisms (2 papers). Amy DeMicco collaborates with scholars based in United States, Sweden and Slovakia. Amy DeMicco's co-authors include Keith R. Cooper, Lori A. White, Jason R. Richardson, Craig H. Bassing, Katherine Yang‐Iott, Outi Vaarala, Oskar Hällgren, Bu Yin, Xiaohong Zhou and Lisa Öberg and has published in prestigious journals such as The Journal of Immunology, PLoS ONE and Cancer Research.

In The Last Decade

Amy DeMicco

12 papers receiving 286 citations

Peers

Amy DeMicco
Amy DeMicco
Citations per year, relative to Amy DeMicco Amy DeMicco (= 1×) peers Ashwini S. Phadnis-Moghe

Countries citing papers authored by Amy DeMicco

Since Specialization
Citations

This map shows the geographic impact of Amy DeMicco's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amy DeMicco with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amy DeMicco more than expected).

Fields of papers citing papers by Amy DeMicco

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amy DeMicco. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amy DeMicco. The network helps show where Amy DeMicco may publish in the future.

Co-authorship network of co-authors of Amy DeMicco

This figure shows the co-authorship network connecting the top 25 collaborators of Amy DeMicco. A scholar is included among the top collaborators of Amy DeMicco based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amy DeMicco. Amy DeMicco is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Kumar, Aditya, Scott P. Simonovich, Christopher J. Kochansky, et al.. (2025). Abstract 5723: XL309 is a potent, selective, and orally bioavailable USP1 inhibitor active as monotherapy and in combination with PARP inhibitors or irinotecan. Cancer Research. 85(8_Supplement_1). 5723–5723. 1 indexed citations
2.
Dumont, Céline, Ulf Sivars, Lina Odqvist, et al.. (2020). A MALT1 inhibitor suppresses human myeloid DC, effector T-cell and B-cell responses and retains Th1/regulatory T-cell homeostasis. PLoS ONE. 15(9). e0222548–e0222548. 18 indexed citations
3.
Hällgren, Oskar, Lisa Öberg, Amy DeMicco, et al.. (2018). Bronchial extracellular matrix from COPD patients induces altered gene expression in repopulated primary human bronchial epithelial cells. Scientific Reports. 8(1). 3502–3502. 27 indexed citations
4.
Malhotra, Rajneesh, Nisha Kurian, Xiaohong Zhou, et al.. (2017). Altered regulation and expression of genes by BET family of proteins in COPD patients. PLoS ONE. 12(3). e0173115–e0173115. 9 indexed citations
6.
Ehrlich, Lori A., Katherine Yang‐Iott, Amy DeMicco, & Craig H. Bassing. (2015). Somatic inactivation of ATM in hematopoietic cells predisposes mice to cyclin D3 dependent T cell acute lymphoblastic leukemia. Cell Cycle. 14(3). 388–398. 3 indexed citations
7.
DeMicco, Amy, Martin S. Naradikian, Vishal Sindhava, et al.. (2015). B Cell–Intrinsic Expression of the HuR RNA-Binding Protein Is Required for the T Cell–Dependent Immune Response In Vivo. The Journal of Immunology. 195(7). 3449–3462. 24 indexed citations
8.
Vaites, Laura Pontano, Zhaorui Lian, Bu Yin, et al.. (2013). ATM deficiency augments constitutively nuclear cyclin D1-driven genomic instability and lymphomagenesis. Oncogene. 33(1). 129–133. 11 indexed citations
9.
DeMicco, Amy, Katherine Yang‐Iott, & Craig H. Bassing. (2013). Somatic inactivation of Tp53 in hematopoietic stem cells or thymocytes predisposes mice to thymic lymphomas with clonal translocations. Cell Cycle. 12(20). 3307–3316. 7 indexed citations
10.
DeMicco, Amy, Julie Horowitz, Bu Yin, et al.. (2011). Tp53 deletion in B lineage cells predisposes mice to lymphomas with oncogenic translocations. Oncogene. 30(47). 4757–4764. 22 indexed citations
11.
White, Lori A., Keith R. Cooper, Amy DeMicco, & Jason R. Richardson. (2010). Using the zebrafish (Danio rerio) model to investigate changes in gene expression and behavior following developmental exposure to pyrethroid pesticides. Neurotoxicology and Teratology. 32(4). 502–503. 1 indexed citations
12.
DeMicco, Amy, Keith R. Cooper, Jason R. Richardson, & Lori A. White. (2009). Developmental Neurotoxicity of Pyrethroid Insecticides in Zebrafish Embryos. Toxicological Sciences. 113(1). 177–186. 161 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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