Amnon Sharir

3.3k total citations
33 papers, 2.6k citations indexed

About

Amnon Sharir is a scholar working on Molecular Biology, Orthopedics and Sports Medicine and Genetics. According to data from OpenAlex, Amnon Sharir has authored 33 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 9 papers in Orthopedics and Sports Medicine and 7 papers in Genetics. Recurrent topics in Amnon Sharir's work include Bone health and osteoporosis research (6 papers), Bone Metabolism and Diseases (4 papers) and Bone and Dental Protein Studies (4 papers). Amnon Sharir is often cited by papers focused on Bone health and osteoporosis research (6 papers), Bone Metabolism and Diseases (4 papers) and Bone and Dental Protein Studies (4 papers). Amnon Sharir collaborates with scholars based in Israel, United States and Japan. Amnon Sharir's co-authors include Elazar Zelzer, Steve Weiner, Julia Mahamid, Lia Addadi, Ron Shahar, Einat Blitz, Sergey Viukov, Haruhiko Akiyama, Randall S. Johnson and Meir M. Barak and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Amnon Sharir

33 papers receiving 2.5k citations

Peers

Amnon Sharir

OSM

RHEUM

SURGE

Damian C. Genetos United States

Yasuyuki Sasano Japan

Christopher S. Adams United States

Norbert Laroche France

Diane Hu United States

Alesha B. Castillo United States

Simon C.F. Rawlinson United Kingdom

Karen Cox United States

Mario Raspanti Italy

Benjamin Gantenbein Switzerland

Damian C. Genetos United States

Amnon Sharir

1.4k ×1.5

771 ×1.3

355 ×0.7

OSM

318 ×0.7

RHEUM

486 ×1.2

SURGE

Citations per year, relative to Amnon Sharir Amnon Sharir (= 1×) peers Damian C. Genetos

Countries citing papers authored by Amnon Sharir

Since Specialization

Citations

This map shows the geographic impact of Amnon Sharir's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amnon Sharir with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amnon Sharir more than expected).

Fields of papers citing papers by Amnon Sharir

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amnon Sharir. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amnon Sharir. The network helps show where Amnon Sharir may publish in the future.

Co-authorship network of co-authors of Amnon Sharir

This figure shows the co-authorship network connecting the top 25 collaborators of Amnon Sharir. A scholar is included among the top collaborators of Amnon Sharir based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amnon Sharir. Amnon Sharir is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Lo, Megan, Amnon Sharir, Michael Paul, et al.. (2022). CNPY4 inhibits the Hedgehog pathway by modulating membrane sterol lipids. Nature Communications. 13(1). 2407–2407. 6 indexed citations

Wong, Jasmine C., Pedro A. Pérez–Mancera, Joaquím Grego‐Bessa, et al.. (2020). KrasP34R and KrasT58I mutations induce distinct RASopathy phenotypes in mice. JCI Insight. 5(21). 8 indexed citations

Sharir, Amnon, Pauline Marangoni, Rapolas Žilionis, et al.. (2019). A large pool of actively cycling progenitors orchestrates self-renewal and injury repair of an ectodermal appendage. Nature Cell Biology. 21(9). 1102–1112. 62 indexed citations

Ransom, Ryan C., Ava C. Carter, Ankit Salhotra, et al.. (2018). Mechanoresponsive stem cells acquire neural crest fate in jaw regeneration. Nature. 563(7732). 514–521. 133 indexed citations

Goodwin, Alice F., William E. Tidyman, Andrew H. Jheon, et al.. (2013). Abnormal Ras signaling in Costello syndrome (CS) negatively regulates enamel formation. Human Molecular Genetics. 23(3). 682–692. 30 indexed citations

Blitz, Einat, Amnon Sharir, Haruhiko Akiyama, & Elazar Zelzer. (2013). Tendon-bone attachment unit is formed modularly by a distinct pool of Scx- and Sox9-positive progenitors. Development. 140(13). 2680–2690. 207 indexed citations

Sharir, Amnon, Joshua Milgram, Gal Dubnov‐Raz, Elazar Zelzer, & Ron Shahar. (2012). A temporary decrease in mineral density in perinatal mouse long bones. Bone. 52(1). 197–205. 5 indexed citations

Sharir, Amnon, et al.. (2011). High Resolution 3D Imaging of <em>Ex-Vivo</em> Biological Samples by Micro CT. Journal of Visualized Experiments. 1 indexed citations

Sharir, Amnon, et al.. (2011). High Resolution 3D Imaging of <em>Ex-Vivo</em> Biological Samples by Micro CT. Journal of Visualized Experiments. 12 indexed citations

10.

Mahamid, Julia, Amnon Sharir, Dvir Gur, et al.. (2011). Bone mineralization proceeds through intracellular calcium phosphate loaded vesicles: A cryo-electron microscopy study. Journal of Structural Biology. 174(3). 527–535. 216 indexed citations

11.

Sharir, Amnon & Elazar Zelzer. (2011). Tendon Homeostasis: The Right Pull. Current Biology. 21(12). R472–R474. 20 indexed citations

12.

Sharir, Amnon, David Israeli, Joshua Milgram, et al.. (2011). The canine baculum: The structure and mechanical properties of an unusual bone. Journal of Structural Biology. 175(3). 451–456. 18 indexed citations

13.

Weißenböck, Martina, et al.. (2010). Mice lacking the orphan receptor ror1 have distinct skeletal abnormalities and are growth retarded. Developmental Dynamics. 239(8). 2266–2277. 32 indexed citations

14.

Vandoorne, Katrien, Jeremy F. Magland, Vicki Plaks, et al.. (2010). Bone vascularization and trabecular bone formation are mediated by PKBalpha/Akt1 in a gene‐dosage‐dependent manner: In vivo and ex vivo MRI. Magnetic Resonance in Medicine. 64(1). 54–64. 13 indexed citations

15.

Blitz, Einat, Sergey Viukov, Amnon Sharir, et al.. (2009). Bone Ridge Patterning during Musculoskeletal Assembly Is Mediated through SCX Regulation of Bmp4 at the Tendon-Skeleton Junction. Developmental Cell. 17(6). 861–873. 225 indexed citations

16.

Shipov, Anna, Amnon Sharir, Elazar Zelzer, et al.. (2009). The influence of severe prolonged exercise restriction on the mechanical and structural properties of bone in an avian model. The Veterinary Journal. 183(2). 153–160. 46 indexed citations

17.

Kahn, Joy, Yulia Shwartz, Einat Blitz, et al.. (2009). Muscle Contraction Is Necessary to Maintain Joint Progenitor Cell Fate. Developmental Cell. 16(5). 734–743. 191 indexed citations

18.

Shivtiel, Shoham, Órit Kollet, Kfir Lapid, et al.. (2008). CD45 regulates retention, motility, and numbers of hematopoietic progenitors, and affects osteoclast remodeling of metaphyseal trabecules. The Journal of Experimental Medicine. 205(10). 2381–2395. 81 indexed citations

19.

Barak, Meir M., Amnon Sharir, & Ron Shahar. (2008). Optical metrology methods for mechanical testing of whole bones. The Veterinary Journal. 180(1). 7–14. 14 indexed citations

20.

Sharir, Amnon, Meir M. Barak, & Ron Shahar. (2007). Whole bone mechanics and mechanical testing. The Veterinary Journal. 177(1). 8–17. 106 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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