Alison Michels

442 total citations
17 papers, 301 citations indexed

About

Alison Michels is a scholar working on Hematology, Internal Medicine and Immunology. According to data from OpenAlex, Alison Michels has authored 17 papers receiving a total of 301 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Hematology, 5 papers in Internal Medicine and 4 papers in Immunology. Recurrent topics in Alison Michels's work include Platelet Disorders and Treatments (7 papers), Blood Coagulation and Thrombosis Mechanisms (7 papers) and Venous Thromboembolism Diagnosis and Management (5 papers). Alison Michels is often cited by papers focused on Platelet Disorders and Treatments (7 papers), Blood Coagulation and Thrombosis Mechanisms (7 papers) and Venous Thromboembolism Diagnosis and Management (5 papers). Alison Michels collaborates with scholars based in Canada, United States and Netherlands. Alison Michels's co-authors include David Lillicrap, Laura L. Swystun, Kate Nesbitt, Kenichi Ogiwara, Wilma M. Hopman, Jeffrey Mewburn, Paula James, Julie Grabell, Patricia C. Liaw and Travis J. Gould and has published in prestigious journals such as SHILAP Revista de lepidopterología, Blood and Arteriosclerosis Thrombosis and Vascular Biology.

In The Last Decade

Alison Michels

17 papers receiving 299 citations

Peers

Alison Michels

HEMAT

IMMUN

IM

MB

PRM

Elizabeth H. Cull United States

Christina C. Rolling Germany

Eleni E Ladikou United Kingdom

Catherine Trichet France

Alessandra Borchiellini Italy

M. S. Enayat United Kingdom

Camila Masias United States

Guillaume Mourey France

Jibran Durrani United States

G Geissler Germany

Elizabeth H. Cull United States

Alison Michels

117 ×0.7

HEMAT

48 ×0.5

IMMUN

21 ×0.4

IM

101 ×2.1

MB

16 ×0.4

PRM

Citations per year, relative to Alison Michels Alison Michels (= 1×) peers Elizabeth H. Cull

Countries citing papers authored by Alison Michels

Since Specialization

Citations

This map shows the geographic impact of Alison Michels's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alison Michels with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alison Michels more than expected).

Fields of papers citing papers by Alison Michels

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alison Michels. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alison Michels. The network helps show where Alison Michels may publish in the future.

Co-authorship network of co-authors of Alison Michels

This figure shows the co-authorship network connecting the top 25 collaborators of Alison Michels. A scholar is included among the top collaborators of Alison Michels based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alison Michels. Alison Michels is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

1.

Shaw, Joseph R., Stephan Nopp, Benedicte Stavik, et al.. (2024). Thrombosis, Translational Medicine, and Biomarker Research: Moving the Needle. Journal of the American Heart Association. 14(1). e038782–e038782. 5 indexed citations

2.

Michels, Alison, et al.. (2024). A narrative review of the role of endoscopically assisted in situ bypass in the modern era of limb salvage vascular bypass. 2. 100078–100078. 1 indexed citations

3.

Swystun, Laura L., Alison Michels, & David Lillicrap. (2023). The contribution of the sinusoidal endothelial cell receptors CLEC4M, stabilin-2, and SCARA5 to VWF–FVIII clearance in thrombosis and hemostasis. Journal of Thrombosis and Haemostasis. 21(8). 2007–2019. 7 indexed citations

4.

Hindmarch, Charles C.T., Kate Nesbitt, Alison Michels, et al.. (2023). The mechanistic and structural role of von Willebrand factor in endotoxemia-enhanced deep vein thrombosis in mice. Journal of Thrombosis and Haemostasis. 21(3). 586–598. 4 indexed citations

5.

Michels, Alison, Laura L. Swystun, Orla Rawley, et al.. (2021). Stabilin‐2 deficiency increases thrombotic burden and alters the composition of venous thrombi in a mouse model. Journal of Thrombosis and Haemostasis. 19(10). 2440–2453. 10 indexed citations

6.

Michels, Alison, et al.. (2021). Role of von Willebrand factor in venous thromboembolic disease. SHILAP Revista de lepidopterología. 3. 17–29. 17 indexed citations

7.

Michels, Alison, Jeffrey Mewburn, Kate Nesbitt, et al.. (2020). von Willebrand Factor Is a Critical Mediator of Deep Vein Thrombosis in a Mouse Model of Diet-Induced Obesity. Arteriosclerosis Thrombosis and Vascular Biology. 40(12). 2860–2874. 22 indexed citations

8.

Swystun, Laura L., Kenichi Ogiwara, Jesse D. Lai, et al.. (2019). The scavenger receptor SCARA5 is an endocytic receptor for von Willebrand factor expressed by littoral cells in the human spleen. Journal of Thrombosis and Haemostasis. 17(8). 1384–1396. 19 indexed citations

9.

Desch, Karl C., Ayse Bilge Ozel, Matthew Halvorsen, et al.. (2017). Exome Sequencing Studies Identify Mutations in STAB2 As a Genetic Risk for Venous Thromboembolic Disease. Blood. 130. 457–457. 4 indexed citations

10.

Michels, Alison, et al.. (2017). Investigating von Willebrand Factor Pathophysiology Using a Flow Chamber Model of von Willebrand Factor-platelet String Formation. Journal of Visualized Experiments. 5 indexed citations

11.

Michels, Alison, et al.. (2017). Investigating von Willebrand Factor Pathophysiology Using a Flow Chamber Model of von Willebrand Factor-platelet String Formation. Journal of Visualized Experiments. 5 indexed citations

12.

Ogiwara, Kenichi, Alison Michels, Wilma M. Hopman, et al.. (2016). Aging and ABO blood type influence von Willebrand factor and factor VIII levels through interrelated mechanisms. Journal of Thrombosis and Haemostasis. 14(5). 953–963. 93 indexed citations

13.

Michels, Alison, Jeffrey Mewburn, Kate Nesbitt, et al.. (2016). Histones link inflammation and thrombosis through the induction of Weibel–Palade body exocytosis. Journal of Thrombosis and Haemostasis. 14(11). 2274–2286. 79 indexed citations

14.

Michels, Alison, et al.. (2014). Histones Induce Endothelial Von Willebrand Factor Release and Subsequent Platelet Capture in In Vitro and In Vivo Models. Blood. 124(21). 2768–2768. 4 indexed citations

15.

Michels, Alison, et al.. (2014). Age-Related Increases in Plasma Factor VIII and Von Willebrand Factor in a C57BL/6 Mouse Model Are Associated with Increased Factor VIII and Von Willebrand Factor Gene Expression and Reduced Expression of the Clearance Receptor, Stabilin-2. Blood. 124(21). 4228–4228. 4 indexed citations

16.

Michels, Alison, et al.. (2013). Reflections as near‐peer facilitators of an inquiry project for undergraduate anatomy: Successes and challenges from a term of trial‐and‐error. Anatomical Sciences Education. 7(1). 64–70. 12 indexed citations

17.

Mommertz, G., Fragiska Sigala, Tim R. Glowka, et al.. (2007). Differences of venous thromboembolic risks in vascular general and trauma surgery patients.. PubMed. 48(6). 727–33. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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