Alina Simerzin

676 total citations
8 papers, 249 citations indexed

About

Alina Simerzin is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Alina Simerzin has authored 8 papers receiving a total of 249 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Cancer Research and 2 papers in Oncology. Recurrent topics in Alina Simerzin's work include MicroRNA in disease regulation (4 papers), Cancer-related molecular mechanisms research (3 papers) and RNA Interference and Gene Delivery (2 papers). Alina Simerzin is often cited by papers focused on MicroRNA in disease regulation (4 papers), Cancer-related molecular mechanisms research (3 papers) and RNA Interference and Gene Delivery (2 papers). Alina Simerzin collaborates with scholars based in Israel, United States and Germany. Alina Simerzin's co-authors include Hilla Giladi, Eithan Galun, Mila Rivkin, Elina Zorde‐Khvalevsky, Nofar Rosenberg, Christoph Schramm, Johannes Kluwe, Jessica Zucman‐Rossi, Reba Condiotti and Shoshana Shpitzen and has published in prestigious journals such as Gastroenterology, Hepatology and Science Advances.

In The Last Decade

Alina Simerzin

8 papers receiving 246 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Alina Simerzin Israel 6 148 131 58 26 24 8 249
Veera Raghavan Thangapandi Germany 4 166 1.1× 120 0.9× 56 1.0× 18 0.7× 9 0.4× 7 280
K Nakamura Japan 9 264 1.8× 211 1.6× 33 0.6× 11 0.4× 23 1.0× 27 396
Enrico Focaccia Italy 6 202 1.4× 146 1.1× 85 1.5× 11 0.4× 24 1.0× 7 337
Klaudia Klicka Poland 9 165 1.1× 136 1.0× 10 0.2× 23 0.9× 18 0.8× 11 324
Céline Bourgne France 9 110 0.7× 37 0.3× 42 0.7× 12 0.5× 29 1.2× 26 271
Biljana Atanasovska Netherlands 5 107 0.7× 69 0.5× 66 1.1× 27 1.0× 31 1.3× 7 188
Bonnie LaCroix United States 9 200 1.4× 154 1.2× 12 0.2× 34 1.3× 7 0.3× 12 270
Jiangjiang Li China 7 106 0.7× 80 0.6× 22 0.4× 8 0.3× 15 0.6× 10 198

Countries citing papers authored by Alina Simerzin

Since Specialization
Citations

This map shows the geographic impact of Alina Simerzin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alina Simerzin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alina Simerzin more than expected).

Fields of papers citing papers by Alina Simerzin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alina Simerzin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alina Simerzin. The network helps show where Alina Simerzin may publish in the future.

Co-authorship network of co-authors of Alina Simerzin

This figure shows the co-authorship network connecting the top 25 collaborators of Alina Simerzin. A scholar is included among the top collaborators of Alina Simerzin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alina Simerzin. Alina Simerzin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Simerzin, Alina, Rainer H. Köhler, Yoshiko Iwamoto, et al.. (2025). Cell confluency affects p53 dynamics in response to DNA damage. Molecular Biology of the Cell. 36(6). br16–br16. 1 indexed citations
2.
Lü, Dan, Bryon Drown, Alina Simerzin, et al.. (2024). Temporal regulation of gene expression through integration of p53 dynamics and modifications. Science Advances. 10(43). eadp2229–eadp2229. 1 indexed citations
3.
Simerzin, Alina, Elina Zorde‐Khvalevsky, Mila Rivkin, et al.. (2021). The lncRNA H19-Derived MicroRNA-675 Promotes Liver Necroptosis by Targeting FADD. Cancers. 13(3). 411–411. 40 indexed citations
4.
Rivkin, Mila, Alina Simerzin, Elina Zorde‐Khvalevsky, et al.. (2017). Metabolic Circuit Involving Free Fatty Acids, microRNA 122, and Triglyceride Synthesis in Liver and Muscle Tissues. Gastroenterology. 153(5). 1404–1415. 82 indexed citations
5.
Rivkin, Mila, Alina Simerzin, Elina Zorde‐Khvalevsky, et al.. (2016). Inflammation-Induced Expression and Secretion of MicroRNA 122 Leads to Reduced Blood Levels of Kidney-Derived Erythropoietin and Anemia. Gastroenterology. 151(5). 999–1010.e3. 51 indexed citations
6.
Simerzin, Alina, Elina Zorde‐Khvalevsky, Mila Rivkin, et al.. (2016). The liver‐specific microRNA‐122*, the complementary strand of microRNA‐122, acts as a tumor suppressor by modulating the p53/mouse double minute 2 homolog circuitry. Hepatology. 64(5). 1623–1636. 41 indexed citations
7.
Condiotti, Reba, Daniel Goldenberg, Hilla Giladi, et al.. (2013). Transduction of Fetal Mice With a Feline Lentiviral Vector Induces Liver Tumors Which Exhibit an E2F Activation Signature. Molecular Therapy. 22(1). 59–68. 11 indexed citations
8.
Grinshpun, Albert, Reba Condiotti, Simon N. Waddington, et al.. (2010). Neonatal Gene Therapy of Glycogen Storage Disease Type Ia Using a Feline Immunodeficiency Virus–based Vector. Molecular Therapy. 18(9). 1592–1598. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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