Ali Zekavat

820 total citations
20 papers, 657 citations indexed

About

Ali Zekavat is a scholar working on Molecular Biology, Genetics and Immunology. According to data from OpenAlex, Ali Zekavat has authored 20 papers receiving a total of 657 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 8 papers in Genetics and 5 papers in Immunology. Recurrent topics in Ali Zekavat's work include Bacterial Genetics and Biotechnology (8 papers), Bacillus and Francisella bacterial research (5 papers) and Lipid Membrane Structure and Behavior (4 papers). Ali Zekavat is often cited by papers focused on Bacterial Genetics and Biotechnology (8 papers), Bacillus and Francisella bacterial research (5 papers) and Lipid Membrane Structure and Behavior (4 papers). Ali Zekavat collaborates with scholars based in United States and Sweden. Ali Zekavat's co-authors include Bruce J. Shenker, Kathleen Boesze‐Battaglia, Donald R. Demuth, Terry McKay, Mensur Dlakić, Edward T. Lally, Lisa Walker, Noboru Yamaguchi, Irving M. Shapiro and Linda L. Otis and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Immunology and International Journal of Molecular Sciences.

In The Last Decade

Ali Zekavat

19 papers receiving 644 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ali Zekavat United States 14 284 146 137 133 132 20 657
Yoko Ueno Japan 6 152 0.5× 52 0.4× 121 0.9× 97 0.7× 116 0.9× 9 383
Riccardo Guidi Sweden 10 276 1.0× 169 1.2× 78 0.6× 24 0.2× 108 0.8× 12 658
Lina Guerra Sweden 8 286 1.0× 87 0.6× 112 0.8× 23 0.2× 145 1.1× 8 535
Weerayuth Kittichotirat Thailand 18 233 0.8× 39 0.3× 96 0.7× 174 1.3× 40 0.3× 50 693
Alice C. L. Len Australia 9 271 1.0× 43 0.3× 50 0.4× 108 0.8× 63 0.5× 9 546
RP Darveau United States 6 115 0.4× 147 1.0× 29 0.2× 89 0.7× 63 0.5× 10 376
Mingsong Kang Canada 10 344 1.2× 98 0.7× 85 0.6× 26 0.2× 65 0.5× 25 587
Eric Oswald France 4 152 0.5× 26 0.2× 68 0.5× 58 0.4× 87 0.7× 4 369
Anders Frisk Sweden 13 414 1.5× 57 0.4× 102 0.7× 23 0.2× 235 1.8× 23 717
Patrick Burr United States 5 242 0.9× 105 0.7× 33 0.2× 26 0.2× 83 0.6× 6 527

Countries citing papers authored by Ali Zekavat

Since Specialization
Citations

This map shows the geographic impact of Ali Zekavat's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ali Zekavat with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ali Zekavat more than expected).

Fields of papers citing papers by Ali Zekavat

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ali Zekavat. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ali Zekavat. The network helps show where Ali Zekavat may publish in the future.

Co-authorship network of co-authors of Ali Zekavat

This figure shows the co-authorship network connecting the top 25 collaborators of Ali Zekavat. A scholar is included among the top collaborators of Ali Zekavat based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ali Zekavat. Ali Zekavat is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Boesze‐Battaglia, Kathleen, Gary H. Cohen, Paul Bates, et al.. (2024). Cellugyrin (synaptogyrin-2) dependent pathways are used by bacterial cytolethal distending toxin and SARS-CoV-2 virus to gain cell entry. Frontiers in Cellular and Infection Microbiology. 14. 1334224–1334224.
3.
Shenker, Bruce J., et al.. (2022). Aggregatibacter actinomycetemcomitans Cytolethal Distending Toxin-Induces Cell Cycle Arrest in a Glycogen Synthase Kinase (GSK)-3-Dependent Manner in Oral Keratinocytes. International Journal of Molecular Sciences. 23(19). 11831–11831. 8 indexed citations
4.
Boesze‐Battaglia, Kathleen, et al.. (2021). The Active Subunit of the Cytolethal Distending Toxin, CdtB, Derived From Both Haemophilus ducreyi and Campylobacter jejuni Exhibits Potent Phosphatidylinositol-3,4,5-Triphosphate Phosphatase Activity. Frontiers in Cellular and Infection Microbiology. 11. 664221–664221. 14 indexed citations
5.
Boesze‐Battaglia, Kathleen, Anuradha Dhingra, Lisa Walker, Ali Zekavat, & Bruce J. Shenker. (2020). Internalization and Intoxication of Human Macrophages by the Active Subunit of the Aggregatibacter actinomycetemcomitans Cytolethal Distending Toxin Is Dependent Upon Cellugyrin (Synaptogyrin-2). Frontiers in Immunology. 11. 1262–1262. 15 indexed citations
6.
Shenker, Bruce J., Lisa Walker, Ali Zekavat, Robert H. Weiss, & Kathleen Boesze‐Battaglia. (2020). The Cell-Cycle Regulatory Protein p21CIP1/WAF1 Is Required for Cytolethal Distending Toxin (Cdt)-Induced Apoptosis. Pathogens. 9(1). 38–38. 11 indexed citations
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Shenker, Bruce J., Larry P. Walker, Ali Zekavat, & Kathleen Boesze‐Battaglia. (2015). Lymphoid susceptibility to the Aggregatibacter actinomycetemcomitans cytolethal distending toxin is dependent upon baseline levels of the signaling lipid, phosphatidylinositol‐3,4,5‐triphosphate. Molecular Oral Microbiology. 31(1). 33–42. 10 indexed citations
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Shenker, Bruce J., et al.. (2010). Inhibition of mast cell degranulation by a chimeric toxin containing a novel phosphatidylinositol-3,4,5-triphosphate phosphatase. Molecular Immunology. 48(1-3). 203–210. 18 indexed citations
13.
Boesze‐Battaglia, Kathleen, Angela C. Brown, Lisa Walker, et al.. (2009). Cytolethal Distending Toxin-induced Cell Cycle Arrest of Lymphocytes Is Dependent upon Recognition and Binding to Cholesterol. Journal of Biological Chemistry. 284(16). 10650–10658. 63 indexed citations
14.
Shenker, Bruce J., Donald R. Demuth, & Ali Zekavat. (2006). Exposure of Lymphocytes to High Doses of Actinobacillus actinomycetemcomitans Cytolethal Distending Toxin Induces Rapid Onset of Apoptosis-Mediated DNA Fragmentation. Infection and Immunity. 74(4). 2080–2092. 31 indexed citations
15.
Shenker, Bruce J., et al.. (2005). Induction of Cell Cycle Arrest in Lymphocytes by Actinobacillus actinomycetemcomitans Cytolethal Distending Toxin Requires Three Subunits for Maximum Activity. The Journal of Immunology. 174(4). 2228–2234. 52 indexed citations
16.
Boesze‐Battaglia, Kathleen, Terry McKay, Ali Zekavat, et al.. (2005). Cholesterol-rich membrane microdomains mediate cell cycle arrest induced by Actinobacillus actinomycetemcomitans cytolethal-distending toxin. Cellular Microbiology. 8(5). 823–836. 64 indexed citations
17.
Shenker, Bruce J., et al.. (2004). Actinobacillus actinomycetemcomitans Cytolethal Distending Toxin (Cdt): Evidence That the Holotoxin Is Composed of Three Subunits: CdtA, CdtB, and CdtC. The Journal of Immunology. 172(1). 410–417. 57 indexed citations
18.
Zekavat, Ali, et al.. (2004). Treponema denticola immunoinhibitory protein induces irreversible G1 arrest in activated human lymphocytes. Oral Microbiology and Immunology. 19(3). 144–149. 12 indexed citations
19.
Shenker, Bruce J., et al.. (2002). Mercury-Induced Apoptosis in Human Lymphocytes: Caspase Activation Is Linked to Redox Status. Antioxidants and Redox Signaling. 4(3). 379–389. 60 indexed citations
20.
Shenker, Bruce J., et al.. (2001). Induction of Apoptosis in Human T Cells by Actinobacillus actinomycetemcomitans Cytolethal Distending Toxin Is a Consequence of G2 Arrest of the Cell Cycle. The Journal of Immunology. 167(1). 435–441. 93 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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