Alexander N.R. Weber

7.4k total citations · 1 hit paper
99 papers, 4.9k citations indexed

About

Alexander N.R. Weber is a scholar working on Immunology, Molecular Biology and Microbiology. According to data from OpenAlex, Alexander N.R. Weber has authored 99 papers receiving a total of 4.9k indexed citations (citations by other indexed papers that have themselves been cited), including 72 papers in Immunology, 40 papers in Molecular Biology and 15 papers in Microbiology. Recurrent topics in Alexander N.R. Weber's work include Immune Response and Inflammation (41 papers), Inflammasome and immune disorders (17 papers) and Antimicrobial Peptides and Activities (15 papers). Alexander N.R. Weber is often cited by papers focused on Immune Response and Inflammation (41 papers), Inflammasome and immune disorders (17 papers) and Antimicrobial Peptides and Activities (15 papers). Alexander N.R. Weber collaborates with scholars based in Germany, United Kingdom and United States. Alexander N.R. Weber's co-authors include Nicholas J. Gay, Dominik Hartl, Monique Gangloff, Markus P. Radsak, Mary A. Morse, Xiaodi Hu, Y. Tony Ip, Takahiro Tanji, Andriy V. Kubarenko and Zsofia Bittner and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Alexander N.R. Weber

97 papers receiving 4.8k citations

Hit Papers

1 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Neutrophils: Between Host Defence, Immune Modulation, and Tissue Injury

2015 495 citations Alexander N.R. Weber, Dominik Hartl et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Alexander N.R. Weber Germany	36	2.9k	1.6k	565	557	425	99	4.9k
Rui Zhou China	27	1.8k 0.6×	2.8k 1.7×	374 0.7×	644 1.2×	161 0.4×	77	5.1k
Vladimir V. Kravchenko United States	27	2.1k 0.7×	2.0k 1.2×	497 0.9×	133 0.2×	390 0.9×	52	4.7k
Alain Beschin Belgium	40	3.5k 1.2×	1.1k 0.7×	1.6k 2.9×	321 0.6×	152 0.4×	84	5.8k
Michael Rehli Germany	47	3.2k 1.1×	3.9k 2.4×	530 0.9×	200 0.4×	219 0.5×	114	7.4k
Ryan H. Moy United States	14	1.1k 0.4×	2.4k 1.5×	1.9k 3.4×	389 0.7×	140 0.3×	33	4.8k
Andor Pivarcsi Sweden	43	2.2k 0.8×	2.7k 1.7×	550 1.0×	139 0.2×	451 1.1×	76	7.0k
Norbert Reiling Germany	37	2.2k 0.8×	1.7k 1.1×	1.4k 2.4×	92 0.2×	165 0.4×	99	5.3k
Paula Preston‐Hurlburt United States	20	6.0k 2.1×	1.6k 1.0×	1.1k 1.9×	112 0.2×	686 1.6×	36	8.1k
Kenji Kawai Japan	36	1.6k 0.6×	1.2k 0.8×	286 0.5×	71 0.1×	237 0.6×	271	4.4k
Liang Qiao China	30	1.2k 0.4×	1.8k 1.1×	481 0.9×	285 0.5×	38 0.1×	106	3.5k

Countries citing papers authored by Alexander N.R. Weber

Since Specialization

Citations

This map shows the geographic impact of Alexander N.R. Weber's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alexander N.R. Weber with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alexander N.R. Weber more than expected).

Fields of papers citing papers by Alexander N.R. Weber

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alexander N.R. Weber. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alexander N.R. Weber. The network helps show where Alexander N.R. Weber may publish in the future.

Co-authorship network of co-authors of Alexander N.R. Weber

This figure shows the co-authorship network connecting the top 25 collaborators of Alexander N.R. Weber. A scholar is included among the top collaborators of Alexander N.R. Weber based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alexander N.R. Weber. Alexander N.R. Weber is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Weber, Alexander N.R., Róisín M. McManus, Veit Hornung, et al.. (2025). The expanding role of the NLRP3 inflammasome from periodic fevers to therapeutic targets. Nature Immunology. 26(9). 1453–1466. 3 indexed citations

Chang, Tzu-Hsuan, Yamel Cardona Gloria, Didier Le Roy, et al.. (2025). Transkingdom mechanism of MAMP generation by chitotriosidase feeds oligomeric chitin from fungal pathogens and allergens into TLR2-mediated innate immune sensing. Frontiers in Immunology. 16. 1497174–1497174. 1 indexed citations

Campbell, Jack H., Alexander N.R. Weber, Dmitry Volodkin, et al.. (2024). Vaterite-based in situ surface modification and process-dependent biocompatibility of laser sintered polypropylene. Journal of Materials Research and Technology. 32. 3447–3455. 2 indexed citations

Leal, Vinícius Nunes Cordeiro, et al.. (2024). Bruton’s tyrosine kinase (BTK) and matrix metalloproteinase-9 (MMP-9) regulate NLRP3 inflammasome-dependent cytokine and neutrophil extracellular trap responses in primary neutrophils. Journal of Allergy and Clinical Immunology. 155(2). 569–582. 10 indexed citations

Maia, Ana, Yamel Cardona Gloria, Katharina Fuchs, et al.. (2023). Chitin oligomers promote lymphoid innate and adaptive immune cell activation. Journal of Leukocyte Biology. 114(2). 180–186. 6 indexed citations

Weber, Alexander N.R., Ana Tapia‐Abellán, Xiao Liu, et al.. (2022). Effective ex vivo inhibition of cryopyrin-associated periodic syndrome (CAPS)-associated mutant NLRP3 inflammasome by MCC950/CRID3. Lara D. Veeken. 61(10). e299–e313. 11 indexed citations

Kazmierski, Julia, Carina Elsner, Katinka Döhner, et al.. (2022). A Baseline Cellular Antiviral State Is Maintained by cGAS and Its Most Frequent Naturally Occurring Variant rs610913. The Journal of Immunology. 209(3). 535–547. 1 indexed citations

Schwaneck, Eva C., Regina Renner, Hans‐Peter Tony, et al.. (2021). Clonal expansion of large granular lymphocytes in patients with spondyloarthritis and psoriatic arthritis treated with TNFα inhibitors. Rheumatology International. 41(11). 1979–1986. 3 indexed citations

Kumpf, Oliver, Saubashya Sur, Jana Eckert, et al.. (2021). A Genetic Variation of Lipopolysaccharide Binding Protein Affects the Inflammatory Response and Is Associated with Improved Outcome during Sepsis. ImmunoHorizons. 5(12). 972–982. 3 indexed citations

10.

Herster, Franziska, Zsofia Bittner, Nathan K. Archer, et al.. (2020). Neutrophil extracellular trap-associated RNA and LL37 enable self-amplifying inflammation in psoriasis. Nature Communications. 11(1). 105–105. 191 indexed citations

11.

Filho, Miguel Inácio da Silva, Frank Christoph, Shun Lu, et al.. (2019). Epistatic effect of TLR3 and cGAS‐STING‐IKKε‐TBK1‐IFN signaling variants on colorectal cancer risk. Cancer Medicine. 9(4). 1473–1484. 12 indexed citations

12.

Nurjadi, Dennis, Klaus Heeg, Alexander N.R. Weber, & Philipp Zanger. (2018). Toll-like receptor 9 (TLR-9) promotor polymorphisms and gene expression are associated with persistent Staphylococcus aureus nasal carriage. Clinical Microbiology and Infection. 24(11). 1210.e7–1210.e12. 16 indexed citations

13.

Murthy, Pranav, Jennifer L. Miller-Ocuin, Xiaoyan Liang, et al.. (2016). The NLRP3 inflammasome and bruton's tyrosine kinase in platelets co-regulate platelet activation, aggregation, and in vitro thrombus formation. Biochemical and Biophysical Research Communications. 483(1). 230–236. 81 indexed citations

14.

Försti, Asta, Jana Eckert, Jelena Knežević, et al.. (2013). Functional TLR5 Genetic Variants Affect Human Colorectal Cancer Survival. Cancer Research. 73(24). 7232–7242. 50 indexed citations

15.

Knežević, Jelena, Dinko Pavlinić, William Rose, et al.. (2012). Heterozygous Carriage of a Dysfunctional Toll-like Receptor 9 Allele Affects CpG Oligonucleotide Responses in B Cells. Journal of Biological Chemistry. 287(29). 24544–24553. 5 indexed citations

16.

Gdynia, Georg, Martina Keith, Jürgen Kopitz, et al.. (2010). Danger Signaling Protein HMGB1 Induces a Distinct Form of Cell Death Accompanied by Formation of Giant Mitochondria. Cancer Research. 70(21). 8558–8568. 44 indexed citations

17.

George, Julie, Andriy V. Kubarenko, Anna Rautanen, et al.. (2010). MyD88 Adaptor-Like D96N Is a Naturally Occurring Loss-of-Function Variant of TIRAP. The Journal of Immunology. 184(6). 3025–3032. 27 indexed citations

18.

Gangloff, Monique, Ayaluru Murali, Jin Xiong, et al.. (2008). Structural Insight into the Mechanism of Activation of the Toll Receptor by the Dimeric Ligand Spätzle. Journal of Biological Chemistry. 283(21). 14629–14635. 61 indexed citations

19.

Andus, Tilo, K. W. Ecker, Andreas Raedler, et al.. (1998). Low dose oral pH modified release budesonide for maintenance of steroid induced remission in Crohn’s disease. Gut. 42(4). 493–496. 80 indexed citations

20.

Frieß, Helmut, M.W. Büchler, C. Beglinger, et al.. (1993). Low-Dose Octreotide Treatment Is Not Effective in Patients with Advanced Pancreatic Cancer. Pancreas. 8(5). 540–545. 47 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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