Alessandro E. Caccamo

553 total citations
9 papers, 395 citations indexed

About

Alessandro E. Caccamo is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Alessandro E. Caccamo has authored 9 papers receiving a total of 395 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 4 papers in Oncology and 3 papers in Cell Biology. Recurrent topics in Alessandro E. Caccamo's work include Clusterin in disease pathology (4 papers), Aldose Reductase and Taurine (2 papers) and Erythrocyte Function and Pathophysiology (2 papers). Alessandro E. Caccamo is often cited by papers focused on Clusterin in disease pathology (4 papers), Aldose Reductase and Taurine (2 papers) and Erythrocyte Function and Pathophysiology (2 papers). Alessandro E. Caccamo collaborates with scholars based in Italy, United Kingdom and United States. Alessandro E. Caccamo's co-authors include Saverio Bettuzzi, Roberta Alfieri, Mara Bonelli, Angelo F. Borghetti, Kenneth P. Wheeler, Andrea Cavazzoni, Andrea Caporali, Maurizio Scaltriti, P. G. Petronini and Pier Giorgio Petronini and has published in prestigious journals such as The Journal of Physiology, JNCI Journal of the National Cancer Institute and Biochemical and Biophysical Research Communications.

In The Last Decade

Alessandro E. Caccamo

9 papers receiving 386 citations

Peers

Alessandro E. Caccamo
Loyal G. Tillotson United States
Vicki L. Nebes United States
Guo-Li Wang United States
Jung-Joo Hwang United States
Loyal G. Tillotson United States
Alessandro E. Caccamo
Citations per year, relative to Alessandro E. Caccamo Alessandro E. Caccamo (= 1×) peers Loyal G. Tillotson

Countries citing papers authored by Alessandro E. Caccamo

Since Specialization
Citations

This map shows the geographic impact of Alessandro E. Caccamo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alessandro E. Caccamo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alessandro E. Caccamo more than expected).

Fields of papers citing papers by Alessandro E. Caccamo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alessandro E. Caccamo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alessandro E. Caccamo. The network helps show where Alessandro E. Caccamo may publish in the future.

Co-authorship network of co-authors of Alessandro E. Caccamo

This figure shows the co-authorship network connecting the top 25 collaborators of Alessandro E. Caccamo. A scholar is included among the top collaborators of Alessandro E. Caccamo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alessandro E. Caccamo. Alessandro E. Caccamo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Chayka, Olesya, Daisy Corvetta, Michael Dews, et al.. (2009). Clusterin, a Haploinsufficient Tumor Suppressor Gene in Neuroblastomas. JNCI Journal of the National Cancer Institute. 101(9). 663–677. 68 indexed citations
2.
Rizzi, Federica, Alessandro E. Caccamo, L Belloni, & Saverio Bettuzzi. (2009). Clusterin is a short half‐life, poly‐ubiquitinated protein, which controls the fate of prostate cancer cells. Journal of Cellular Physiology. 219(2). 314–323. 44 indexed citations
3.
Schwáb, R., Alessandro E. Caccamo, Saverio Bettuzzi, John Anderson, & Arturo Sala. (2007). B-MYB is hypophosphorylated and resistant to degradation in neuroblastoma: Implications for cell survival. Blood Cells Molecules and Diseases. 39(3). 263–271. 9 indexed citations
4.
Scaltriti, Maurizio, Saverio Bettuzzi, R. M. Sharrard, et al.. (2004). Clusterin overexpression in both malignant and nonmalignant prostate epithelial cells induces cell cycle arrest and apoptosis. British Journal of Cancer. 91(10). 1842–1850. 64 indexed citations
5.
Caccamo, Alessandro E., Maurizio Scaltriti, Andrea Caporali, et al.. (2003). Nuclear Translocation of a Clusterin Isoform Is Associated with Induction of Anoikis in SV40‐Immortalized Human Prostate Epithelial Cells. Annals of the New York Academy of Sciences. 1010(1). 514–519. 35 indexed citations
6.
Alfieri, Roberta, Andrea Cavazzoni, Pier Giorgio Petronini, et al.. (2002). Compatible osmolytes modulate the response of porcine endothelial cells to hypertonicity and protect them from apoptosis. The Journal of Physiology. 540(2). 499–508. 82 indexed citations
7.
Petronini, P. G., Alessandro E. Caccamo, Roberta Alfieri, Mara Bonelli, & A. F. Borghetti. (2001). The effect of heat shock on amino acid transport and cell volume in 3T3 cells. Amino Acids. 20(4). 363–380. 4 indexed citations
8.
Alfieri, Roberta, P. G. Petronini, Mara Bonelli, et al.. (2001). Osmotic Regulation of ATA2 mRNA Expression and Amino Acid Transport System A Activity. Biochemical and Biophysical Research Communications. 283(1). 174–178. 48 indexed citations
9.
Petronini, P. G., Roberta Alfieri, Alessandro E. Caccamo, et al.. (2000). Induction of BGT-1 and amino acid System A transport activities in endothelial cells exposed to hyperosmolarity. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 279(5). R1580–R1589. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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