Aiko Ishii

685 total citations
26 papers, 551 citations indexed

About

Aiko Ishii is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Oncology. According to data from OpenAlex, Aiko Ishii has authored 26 papers receiving a total of 551 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 5 papers in Cellular and Molecular Neuroscience and 5 papers in Oncology. Recurrent topics in Aiko Ishii's work include Protease and Inhibitor Mechanisms (3 papers), Neuropeptides and Animal Physiology (3 papers) and Parkinson's Disease Mechanisms and Treatments (2 papers). Aiko Ishii is often cited by papers focused on Protease and Inhibitor Mechanisms (3 papers), Neuropeptides and Animal Physiology (3 papers) and Parkinson's Disease Mechanisms and Treatments (2 papers). Aiko Ishii collaborates with scholars based in Japan, United States and China. Aiko Ishii's co-authors include Atsushi Nakamura, Junko Sakagami, Makoto Yoshimoto, Takeshi Iwatsubo, Kazuto Kobayashi, Shingo Iwata, Makoto Inui, Takeshi Honda, Yasuharu Oishi and Toshiharu Hayashi and has published in prestigious journals such as Annals of the New York Academy of Sciences, Molecular Biology of the Cell and Life Sciences.

In The Last Decade

Aiko Ishii

25 papers receiving 535 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Aiko Ishii Japan 13 182 132 129 91 52 26 551
Ryoichi Kawamura Japan 16 59 0.3× 177 1.3× 108 0.8× 166 1.8× 29 0.6× 55 828
Nobuhiko Omori Japan 16 112 0.6× 216 1.6× 135 1.0× 63 0.7× 37 0.7× 46 851
Yoshihito Shimazu Japan 15 40 0.2× 202 1.5× 123 1.0× 248 2.7× 63 1.2× 56 665
May C. Ng United States 11 39 0.2× 188 1.4× 83 0.6× 54 0.6× 34 0.7× 20 547
Eloïse Lemarchand United Kingdom 16 98 0.5× 296 2.2× 58 0.4× 41 0.5× 18 0.3× 25 709
H Meixner Germany 13 202 1.1× 197 1.5× 112 0.9× 141 1.5× 24 0.5× 18 674
P Thomsen Denmark 13 164 0.9× 81 0.6× 137 1.1× 109 1.2× 16 0.3× 20 773
Evangelia Kesidou Greece 13 78 0.4× 200 1.5× 98 0.8× 76 0.8× 36 0.7× 45 663
Denisse Sepúlveda Chile 12 51 0.3× 182 1.4× 47 0.4× 196 2.2× 18 0.3× 19 620
Bo Ma China 19 102 0.6× 333 2.5× 71 0.6× 39 0.4× 25 0.5× 56 762

Countries citing papers authored by Aiko Ishii

Since Specialization
Citations

This map shows the geographic impact of Aiko Ishii's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aiko Ishii with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aiko Ishii more than expected).

Fields of papers citing papers by Aiko Ishii

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Aiko Ishii. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aiko Ishii. The network helps show where Aiko Ishii may publish in the future.

Co-authorship network of co-authors of Aiko Ishii

This figure shows the co-authorship network connecting the top 25 collaborators of Aiko Ishii. A scholar is included among the top collaborators of Aiko Ishii based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aiko Ishii. Aiko Ishii is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yamashita, Haruhiro, et al.. (2017). Morphological analysis of patchy thickening and reddish discoloration of active hair growth areas in the skin of New Zealand White rabbits. Journal of Toxicologic Pathology. 30(4). 315–322. 7 indexed citations
2.
3.
Honda, Takeshi, Aiko Ishii, & Makoto Inui. (2013). Regulation of adipocyte differentiation of 3T3-L1 cells by PDZRN3. American Journal of Physiology-Cell Physiology. 304(11). C1091–C1097. 13 indexed citations
4.
Honda, Takeshi, et al.. (2010). PDZRN3 Negatively Regulates BMP-2–induced Osteoblast Differentiation through Inhibition of Wnt Signaling. Molecular Biology of the Cell. 21(18). 3269–3277. 33 indexed citations
5.
Tsuritani, Katsuki, Junko Takeda, Junko Sakagami, et al.. (2009). Cytokine Receptor-Like Factor 1 is Highly Expressed in Damaged Human Knee Osteoarthritic Cartilage and Involved in Osteoarthritis Downstream of TGF-β. Calcified Tissue International. 86(1). 47–57. 28 indexed citations
6.
Nakamura, Misa, Takashi Ozaki, Aiko Ishii, et al.. (2009). Calcitonin induces connective tissue growth factor through ERK1/2 signaling in renal tubular cells. Experimental & Molecular Medicine. 41(5). 307–307. 3 indexed citations
7.
Odani, Hiroko, Aiko Ishii, Atsushi Nakamura, et al.. (2008). Suppression of Renal α‐Dicarbonyl Compounds Generated following Ureteral Obstruction by Kidney‐Specific α‐Dicarbonyl/l‐Xylulose Reductase. Annals of the New York Academy of Sciences. 1126(1). 320–324. 12 indexed citations
8.
Kuwahara, Yusuke, et al.. (2008). Downregulated expression in high IgA (HIGA) mice and the renal protective role of meprinβ. Life Sciences. 82(15-16). 899–908. 6 indexed citations
9.
Ishii, Aiko, Junko Sakagami, Shingo Iwata, et al.. (2007). Accumulation of phosphorylated α‐synuclein in dopaminergic neurons of transgenic mice that express human α‐synuclein. Journal of Neuroscience Research. 85(8). 1819–1825. 69 indexed citations
10.
Nakamura, Atsushi, et al.. (2007). Early induction of osteoactivin expression in rat renal tubular epithelial cells after unilateral ureteral obstruction. Experimental and Toxicologic Pathology. 59(1). 53–59. 19 indexed citations
11.
Ishii, Aiko, et al.. (2007). Molecular Pathological Evaluation of Clusterin in a Rat Model of Unilateral Ureteral Obstruction as a Possible Biomarker of Nephrotoxicity. Toxicologic Pathology. 35(3). 376–382. 30 indexed citations
12.
Ishii, Aiko, Shingo Iwata, Junko Sakagami, et al.. (2006). Selective loss of nigral dopamine neurons induced by overexpression of truncated human α-synuclein in mice. Neurobiology of Aging. 29(4). 574–585. 121 indexed citations
13.
Han, Bo, Misa Nakamura, Gengyin Zhou, et al.. (2006). Calcitonin inhibits invasion of breast cancer cells: Involvement of urokinase-type plasminogen activator (uPA) and uPA receptor. International Journal of Oncology. 28(4). 807–14. 17 indexed citations
14.
Ishii, Aiko, Misa Nakamura, Atsushi Nakamura, et al.. (2006). Expression of Calcitonin Receptor in Rat Mammary Gland during Lactation. Endocrine Journal. 53(3). 317–324. 4 indexed citations
15.
Ishii, Aiko, Yoshikatsu Uematsu, Atsushi Nakamura, et al.. (2005). Transgenic Mice Over-expressing Dicarbonyl/L-xylulose Reductase Gene Crossed with KK-Ay Diabetic Model Mice: An Animal Model for the Metabolism of Renal Carbonyl Compounds. EXPERIMENTAL ANIMALS. 54(5). 385–394. 12 indexed citations
16.
Hayashi, Toshiharu, Aiko Ishii, Satoru Nakai, & Keiko Hasegawa. (2004). Ultrastructure of goblet-cell metaplasia from Clara cell in the allergic asthmatic airway inflammation in a mouse model of asthma in vivo. Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin. 444(1). 66–73. 43 indexed citations
17.
Nunobiki, Osamu, Emiko Taniguchi, Aiko Ishii, et al.. (2003). Significance of hormone receptor status and tumor vessels in normal, hyperplastic and neoplastic endometrium. Pathology International. 53(12). 846–852. 13 indexed citations
18.
Hagiwara, Yuriko, Aiko Ishii, Ikuya Nonaka, Tateki Kikuchi, & Shin’ichi Takeda. (1998). Fiber-type-dependent expression of adenovirus-mediated transgene in mouse skeletal muscle fibers. Acta Neuropathologica. 96(3). 228–232. 2 indexed citations
19.
20.
Ishii, Aiko, Shiro Watanabe, & Hatsuyuki Hamada. (1992). ATP-dependent protease in human placenta. Placenta. 13(4). 343–347. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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