Ahmed Abidi

521 total citations
8 papers, 302 citations indexed

About

Ahmed Abidi is a scholar working on Immunology, Pathology and Forensic Medicine and Oncology. According to data from OpenAlex, Ahmed Abidi has authored 8 papers receiving a total of 302 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology, 3 papers in Pathology and Forensic Medicine and 3 papers in Oncology. Recurrent topics in Ahmed Abidi's work include IL-33, ST2, and ILC Pathways (3 papers), Psoriasis: Treatment and Pathogenesis (2 papers) and Immune Cell Function and Interaction (2 papers). Ahmed Abidi is often cited by papers focused on IL-33, ST2, and ILC Pathways (3 papers), Psoriasis: Treatment and Pathogenesis (2 papers) and Immune Cell Function and Interaction (2 papers). Ahmed Abidi collaborates with scholars based in France, Tunisia and United States. Ahmed Abidi's co-authors include Gaëlle Bériou, Régis Josien, Jérôme C. Martin, Jean‐Christophe Renauld, Ermanno Candolfi, Alexander W. Pfaff, Yusuke Nakamura, Motoko Unoki, J.P. Klein and Julie Brunet and has published in prestigious journals such as The Journal of Immunology, Scientific Reports and Frontiers in Immunology.

In The Last Decade

Ahmed Abidi

8 papers receiving 301 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Ahmed Abidi France	8	151	77	76	64	48	8	302
Valérie Staudt Germany	4	435 2.9×	53 0.7×	20 0.3×	40 0.6×	105 2.2×	4	553
Elaine Huang United States	5	175 1.2×	55 0.7×	21 0.3×	22 0.3×	57 1.2×	11	265
Lisa Kleißl Austria	11	220 1.5×	71 0.9×	37 0.5×	17 0.3×	52 1.1×	19	360
N. Hmida Tunisia	7	249 1.6×	32 0.4×	113 1.5×	9 0.1×	60 1.3×	13	374
Kordula Kautz‐Neu Germany	6	311 2.1×	86 1.1×	38 0.5×	14 0.2×	34 0.7×	7	406
S. Gatault Ireland	8	127 0.8×	51 0.7×	14 0.2×	46 0.7×	48 1.0×	13	311
Goesta Gahrton Sweden	7	57 0.4×	51 0.7×	133 1.8×	34 0.5×	63 1.3×	15	301
Nicolaas E. Aerts Belgium	9	218 1.4×	32 0.4×	13 0.2×	22 0.3×	29 0.6×	11	431
Valérie Hay Canada	6	335 2.2×	29 0.4×	57 0.8×	16 0.3×	62 1.3×	6	411
Harumi Saeki Japan	11	35 0.2×	135 1.8×	40 0.5×	33 0.5×	167 3.5×	37	366

Countries citing papers authored by Ahmed Abidi

Since Specialization

Citations

This map shows the geographic impact of Ahmed Abidi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ahmed Abidi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ahmed Abidi more than expected).

Fields of papers citing papers by Ahmed Abidi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ahmed Abidi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ahmed Abidi. The network helps show where Ahmed Abidi may publish in the future.

Co-authorship network of co-authors of Ahmed Abidi

This figure shows the co-authorship network connecting the top 25 collaborators of Ahmed Abidi. A scholar is included among the top collaborators of Ahmed Abidi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ahmed Abidi. Ahmed Abidi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Mérieau, Emmanuel, Gaëlle Bériou, Tuan Huy Nguyen, et al.. (2021). Cystathionine‐gamma‐lyase overexpression in T cells enhances antitumor effect independently of cysteine autonomy. Cancer Science. 112(5). 1723–1734. 14 indexed citations

2.

Abidi, Ahmed, Gaëlle Bériou, Laurence Bouchet‐Delbos, et al.. (2020). Characterization of Rat ILCs Reveals ILC2 as the Dominant Intestinal Subset. Frontiers in Immunology. 11. 255–255. 14 indexed citations

3.

Heim, Xavier, D. S. Akram, Asma Gati, et al.. (2019). TH17 cells expressing CD146 are significantly increased in patients with Systemic sclerosis. Scientific Reports. 9(1). 17721–17721. 24 indexed citations

4.

Martin, Jérôme C., Kerstin Wolk, Gaëlle Bériou, et al.. (2017). Limited Presence of IL-22 Binding Protein, a Natural IL-22 Inhibitor, Strengthens Psoriatic Skin Inflammation. The Journal of Immunology. 198(9). 3671–3678. 56 indexed citations

5.

Blancato, Jan, David Goerlitz, Md. Sajedul Islam, et al.. (2016). Circulating Cell-free miRNA Expression and its Association with Clinicopathologic Features in Inflammatory and Non-Inflammatory Breast Cancer. Asian Pacific Journal of Cancer Prevention. 17(4). 1801–1810. 10 indexed citations

6.

Martin, Jérôme C., Gaëlle Bériou, M Heslan, et al.. (2015). IL-22BP is produced by eosinophils in human gut and blocks IL-22 protective actions during colitis. Mucosal Immunology. 9(2). 539–549. 76 indexed citations

7.

Bougatef, Karim, Amel Mézlini, Ahmed Abidi, et al.. (2014). Significant association between IL23R and IL17F polymorphisms and clinical features of colorectal cancer. Immunology Letters. 158(1-2). 189–194. 33 indexed citations

8.

Brunet, Julie, Alexander W. Pfaff, Ahmed Abidi, et al.. (2008). Toxoplasma gondii exploits UHRF1 and induces host cell cycle arrest at G2 to enable its proliferation. Cellular Microbiology. 10(4). 908–920. 75 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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