Ágnes Végh

1.8k total citations
69 papers, 1.3k citations indexed

About

Ágnes Végh is a scholar working on Pathology and Forensic Medicine, Cardiology and Cardiovascular Medicine and Emergency Medicine. According to data from OpenAlex, Ágnes Végh has authored 69 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 45 papers in Pathology and Forensic Medicine, 45 papers in Cardiology and Cardiovascular Medicine and 23 papers in Emergency Medicine. Recurrent topics in Ágnes Végh's work include Cardiac Ischemia and Reperfusion (45 papers), Cardiac electrophysiology and arrhythmias (39 papers) and Cardiac Arrest and Resuscitation (23 papers). Ágnes Végh is often cited by papers focused on Cardiac Ischemia and Reperfusion (45 papers), Cardiac electrophysiology and arrhythmias (39 papers) and Cardiac Arrest and Resuscitation (23 papers). Ágnes Végh collaborates with scholars based in Hungary, United Kingdom and Finland. Ágnes Végh's co-authors include James R. Parratt, Julius Gy. Papp, L. Szekeres, Éva Udvary, Mária Kovács, Elmar Krause, Julius Gyula Papp, K. Geisen, György Seprényi and Orsolya Nagy and has published in prestigious journals such as PLoS ONE, International Journal of Molecular Sciences and Annals of the New York Academy of Sciences.

In The Last Decade

Ágnes Végh

68 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ágnes Végh Hungary 20 811 663 445 289 284 69 1.3k
Francesca Tullio Italy 20 608 0.7× 290 0.4× 371 0.8× 497 1.7× 206 0.7× 39 1.3k
Mahiko Goto Japan 13 1.1k 1.4× 378 0.6× 672 1.5× 247 0.9× 133 0.5× 15 1.3k
Petra Gres Germany 20 921 1.1× 665 1.0× 477 1.1× 721 2.5× 247 0.9× 29 1.7k
J. Craig Hartman United States 15 323 0.4× 364 0.5× 170 0.4× 146 0.5× 153 0.5× 30 899
Akihito Tsuchida Japan 23 1.4k 1.7× 566 0.9× 814 1.8× 342 1.2× 141 0.5× 42 1.7k
H PIPER Germany 11 565 0.7× 361 0.5× 257 0.6× 382 1.3× 118 0.4× 21 993
Joseph Loufouat France 15 996 1.2× 403 0.6× 584 1.3× 442 1.5× 75 0.3× 22 1.5k
Liliane Tariosse France 14 416 0.5× 457 0.7× 141 0.3× 355 1.2× 151 0.5× 23 1.0k
Sandrine Pons France 18 284 0.4× 268 0.4× 185 0.4× 333 1.2× 98 0.3× 39 902
Christian Mouas France 20 482 0.6× 542 0.8× 255 0.6× 412 1.4× 105 0.4× 44 1.6k

Countries citing papers authored by Ágnes Végh

Since Specialization
Citations

This map shows the geographic impact of Ágnes Végh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ágnes Végh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ágnes Végh more than expected).

Fields of papers citing papers by Ágnes Végh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ágnes Végh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ágnes Végh. The network helps show where Ágnes Végh may publish in the future.

Co-authorship network of co-authors of Ágnes Végh

This figure shows the co-authorship network connecting the top 25 collaborators of Ágnes Végh. A scholar is included among the top collaborators of Ágnes Végh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ágnes Végh. Ágnes Végh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Nagy, László, Piero Pollesello, Heimo Haikala, et al.. (2016). ORM-3819 promotes cardiac contractility through Ca2+ sensitization in combination with selective PDE III inhibition, a novel approach to inotropy. European Journal of Pharmacology. 775. 120–129. 4 indexed citations
2.
3.
Végh, Ágnes, et al.. (2013). Regulation of gap junctions by nitric oxide influences the generation of arrhythmias resulting from acute ischemia and reperfusion in vivo. Frontiers in Pharmacology. 4. 76–76. 10 indexed citations
4.
Kiss, Attila, et al.. (2011). Is there a trigger role of peroxynitrite in the anti-arrhythmic effect of ischaemic preconditioning and peroxynitrite infusion?. European Journal of Pharmacology. 667(1-3). 306–313. 5 indexed citations
5.
Kiss, Attila, et al.. (2010). The role of nitric oxide, superoxide and peroxynitrite in the anti‐arrhythmic effects of preconditioning and peroxynitrite infusion in anaesthetized dogs. British Journal of Pharmacology. 160(5). 1263–1272. 18 indexed citations
6.
Kovács, Mária, et al.. (2010). Changes in gene expression following cardiac pacing-induced delayed cardioprotection in the canine heart. Acta Biologica Hungarica. 61(4). 434–448. 4 indexed citations
7.
Papp, Rita, et al.. (2009). Modulation of gap junctions by nitric oxide contributes to the anti‐arrhythmic effect of sodium nitroprusside?. British Journal of Pharmacology. 156(5). 786–793. 19 indexed citations
8.
Nagy, Orsolya, et al.. (2005). Nitric oxide involvement in the delayed antiarrhythmic effect of treadmill exercise in dogs. Life Sciences. 77(16). 1960–1971. 14 indexed citations
9.
10.
Végh, Ágnes & James R. Parratt. (2002). The role of mitochondrial KATP channels in antiarrhythmic effects of ischaemic preconditioning in dogs. British Journal of Pharmacology. 137(7). 1107–1115. 30 indexed citations
11.
Végh, Ágnes, et al.. (2000). Atrial Natriuretic Peptide Reduces the Severe Consequences of Coronary Artery Occlusion in Anaesthetized Dogs. Cardiovascular Drugs and Therapy. 14(5). 471–479. 16 indexed citations
12.
Végh, Ágnes, et al.. (1999). Delayed protection against ventricular arrhythmias by monophosphoryl lipid-A in a canine model of ischaemia and reperfusion. European Journal of Pharmacology. 382(2). 81–90. 4 indexed citations
13.
Végh, Ágnes, et al.. (1996). Nicorandil suppressed ventricular arrhythmias in a canine model of myocardial ischaemia. European Journal of Pharmacology. 305(1-3). 163–168. 20 indexed citations
14.
Geisen, K., Ágnes Végh, Elmar Krause, & Julius Gyula Papp. (1996). Cardiovascular Effects of Conventional Sulfonylureas and Glimepiride. Hormone and Metabolic Research. 28(9). 496–507. 66 indexed citations
15.
Wilson, Susan, Song Wu, T Ravíngerová, et al.. (1996). Delayed cardioprotection is associated with the sub-cellular relocalisation of ventricular protein kinase C?, but not p42/44MAPK. Molecular and Cellular Biochemistry. 160-161(1). 225–230. 26 indexed citations
16.
Végh, Ágnes & Julius Gy. Papp. (1996). Haemodynamic and other effects of sulphonylurea drugs on the heart. Diabetes Research and Clinical Practice. 31. S43–S53. 16 indexed citations
17.
Udvary, Éva, Julius Gy. Papp, & Ágnes Végh. (1995). Cardiovascular effects of the calcium sensitizer, levosimendan, in heart failure induced by rapid pacing in the presence of aortic constriction. British Journal of Pharmacology. 114(3). 656–661. 33 indexed citations
18.
Végh, Ágnes, Julius Gy. Papp, & James R. Parratt. (1994). Prevention by dexamethasone of the marked antiarrhythmic effects of preconditioning induced 20 h after rapid cardiac pacing. British Journal of Pharmacology. 113(4). 1081–1082. 67 indexed citations
19.
Végh, Ágnes, Julius Gy. Papp, L. Szekeres, & James R. Parratt. (1992). The local intracoronary administration of methylene blue prevents the pronounced antiarrhythmic effect of ischaemic preconditioning. British Journal of Pharmacology. 107(4). 910–911. 47 indexed citations
20.
Végh, Ágnes, L. Szekeres, & James R. Parratt. (1991). Local intracoronary infusions of bradykinin profoundly reduce the severity of ischaemia‐induced arrhythmias in anaesthetized dogs. British Journal of Pharmacology. 104(2). 294–295. 88 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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