Ada Weinstock

1.6k total citations
21 papers, 1.1k citations indexed

About

Ada Weinstock is a scholar working on Immunology, Molecular Biology and Epidemiology. According to data from OpenAlex, Ada Weinstock has authored 21 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Immunology, 9 papers in Molecular Biology and 7 papers in Epidemiology. Recurrent topics in Ada Weinstock's work include Adipokines, Inflammation, and Metabolic Diseases (7 papers), Atherosclerosis and Cardiovascular Diseases (5 papers) and Immune cells in cancer (5 papers). Ada Weinstock is often cited by papers focused on Adipokines, Inflammation, and Metabolic Diseases (7 papers), Atherosclerosis and Cardiovascular Diseases (5 papers) and Immune cells in cancer (5 papers). Ada Weinstock collaborates with scholars based in United States, Israel and Sweden. Ada Weinstock's co-authors include Edward A. Fisher, Kathryn J. Moore, P’ng Loke, Karishma Rahman, Emily J. Brown, Stephen A. Ramsey, Monika Sharma, Hitoo Nishi, Jianhua Liu and Natasha Girgis and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Ada Weinstock

21 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ada Weinstock United States 15 686 426 310 131 114 21 1.1k
Günther Staffler Austria 20 529 0.8× 472 1.1× 169 0.5× 182 1.4× 132 1.2× 29 1.4k
Pontus Dunér Sweden 20 552 0.8× 489 1.1× 184 0.6× 99 0.8× 200 1.8× 44 1.2k
Sheri Kelemen United States 25 473 0.7× 462 1.1× 90 0.3× 97 0.7× 147 1.3× 36 1.1k
Maria Wigren Sweden 21 838 1.2× 258 0.6× 272 0.9× 58 0.4× 95 0.8× 34 1.1k
Ragnar Alm Sweden 23 589 0.9× 250 0.6× 233 0.8× 92 0.7× 174 1.5× 35 1.1k
Karishma Rahman United States 9 645 0.9× 430 1.0× 250 0.8× 53 0.4× 127 1.1× 15 1.1k
Meixiang Yang China 20 503 0.7× 495 1.2× 139 0.4× 152 1.2× 131 1.1× 38 1.2k
Yoshishige Miyabe Japan 19 425 0.6× 362 0.8× 126 0.4× 104 0.8× 77 0.7× 42 1.2k
Guang‐Nian Zhao China 16 233 0.3× 430 1.0× 218 0.7× 59 0.5× 65 0.6× 24 868

Countries citing papers authored by Ada Weinstock

Since Specialization
Citations

This map shows the geographic impact of Ada Weinstock's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ada Weinstock with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ada Weinstock more than expected).

Fields of papers citing papers by Ada Weinstock

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ada Weinstock. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ada Weinstock. The network helps show where Ada Weinstock may publish in the future.

Co-authorship network of co-authors of Ada Weinstock

This figure shows the co-authorship network connecting the top 25 collaborators of Ada Weinstock. A scholar is included among the top collaborators of Ada Weinstock based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ada Weinstock. Ada Weinstock is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Scolaro, Bianca, Emily J. Brown, Marie Petitjean, et al.. (2025). Caloric restriction promotes resolution of atherosclerosis in obese mice, while weight regain accelerates its progression. Journal of Clinical Investigation. 135(18). 2 indexed citations
2.
Yu, Liming, Lin Xu, Haiyan Chu, et al.. (2023). Macrophage-to-endothelial cell crosstalk by the cholesterol metabolite 27HC promotes atherosclerosis in male mice. Nature Communications. 14(1). 4101–4101. 25 indexed citations
3.
Weinstock, Ada, et al.. (2023). The diverse roles of macrophages in metabolic inflammation and its resolution. Frontiers in Cell and Developmental Biology. 11. 1147434–1147434. 19 indexed citations
4.
Zwack, Erin E., Ze Chen, Joseph C. Devlin, et al.. (2022). Staphylococcus aureusinduces a muted host response in human blood that blunts the recruitment of neutrophils. Proceedings of the National Academy of Sciences. 119(31). e2123017119–e2123017119. 19 indexed citations
5.
Heffron, Sean, Ada Weinstock, Bianca Scolaro, et al.. (2020). Platelet‐conditioned media induces an anti‐inflammatory macrophage phenotype through EP4. Journal of Thrombosis and Haemostasis. 19(2). 562–573. 19 indexed citations
6.
Weinstock, Ada, Hernandez Moura Silva, Kathryn J. Moore, Ann Marie Schmidt, & Edward A. Fisher. (2020). Leukocyte Heterogeneity in Adipose Tissue, Including in Obesity. Circulation Research. 126(11). 1590–1612. 45 indexed citations
7.
Sharma, Monika, Martin Schlegel, Milessa Silva Afonso, et al.. (2020). Regulatory T Cells License Macrophage Pro-Resolving Functions During Atherosclerosis Regression. Circulation Research. 127(3). 335–353. 182 indexed citations
8.
9.
Weinstock, Ada, Julio Gallego‐Delgado, Cláudia Gomes, et al.. (2019). Tamoxifen activity against Plasmodium in vitro and in mice. Malaria Journal. 18(1). 378–378. 17 indexed citations
10.
Weinstock, Ada & Edward A. Fisher. (2019). Methods to Study Monocyte and Macrophage Trafficking in Atherosclerosis Progression and Resolution. Methods in molecular biology. 1951. 153–165. 11 indexed citations
11.
12.
Sharma, Monika, Martin Schlegel, Emily J. Brown, et al.. (2019). Netrin-1 Alters Adipose Tissue Macrophage Fate and Function in Obesity. PubMed. 1(2). 41 indexed citations
13.
14.
Rahman, Karishma, Yuliya Vengrenyuk, Stephen A. Ramsey, et al.. (2017). Inflammatory Ly6Chi monocytes and their conversion to M2 macrophages drive atherosclerosis regression. Journal of Clinical Investigation. 127(8). 2904–2915. 256 indexed citations
15.
Peled, Michael, Hitoo Nishi, Ada Weinstock, et al.. (2017). A wild-type mouse-based model for the regression of inflammation in atherosclerosis. PLoS ONE. 12(3). e0173975–e0173975. 39 indexed citations
16.
Gallego‐Delgado, Julio, Upal Basu-Roy, Maureen Ty, et al.. (2016). Angiotensin receptors and β-catenin regulate brain endothelial integrity in malaria. Journal of Clinical Investigation. 126(10). 4016–4029. 56 indexed citations
17.
Weinstock, Ada, Meirav Pevsner‐Fischer, Ziv Porat, Michael Selitrennik, & Dov Zipori. (2015). Cultured Mesenchymal Stem Cells Stimulate an Immune Response by Providing Immune Cells with Toll-Like Receptor 2 Ligand. Stem Cell Reviews and Reports. 11(6). 826–840. 7 indexed citations
18.
Pevsner‐Fischer, Meirav, et al.. (2014). Divergent Levels of LBP and TGFβ1 in Murine MSCs Lead to Heterogenic Response to TLR and Proinflammatory Cytokine Activation. Stem Cell Reviews and Reports. 10(3). 376–388. 11 indexed citations
19.
Ravid, Orly, Ofer Shoshani, Meirav Sela, et al.. (2014). Relative genomic stability of adipose tissue derived mesenchymal stem cells: analysis of ploidy, H19 long non-coding RNA and p53 activity. Stem Cell Research & Therapy. 5(6). 139–139. 19 indexed citations
20.
Weinstock, Ada, et al.. (1994). Pleomorphic carcinoma (spindle and giant cell) of the lung.. PubMed. 43(9). 787–90. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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