Abderrahim Merzak

1.1k total citations
22 papers, 903 citations indexed

About

Abderrahim Merzak is a scholar working on Molecular Biology, Cell Biology and Oncology. According to data from OpenAlex, Abderrahim Merzak has authored 22 papers receiving a total of 903 indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 7 papers in Cell Biology and 6 papers in Oncology. Recurrent topics in Abderrahim Merzak's work include Glycosylation and Glycoproteins Research (7 papers), Cell Adhesion Molecules Research (6 papers) and TGF-β signaling in diseases (5 papers). Abderrahim Merzak is often cited by papers focused on Glycosylation and Glycoproteins Research (7 papers), Cell Adhesion Molecules Research (6 papers) and TGF-β signaling in diseases (5 papers). Abderrahim Merzak collaborates with scholars based in United Kingdom, France and Poland. Abderrahim Merzak's co-authors include Geoffrey J. Pilkington, Shahriar Koochekpour, G. J. Pilkington, Marie‐Paule Fillion, David A. Lawrence, Gajanan V. Sherbet, Philippe Vigier, Omar Benzakour, Harcharan K. Rooprai and Garry J. Rucklidge and has published in prestigious journals such as British Journal of Cancer, International Journal of Cancer and European Journal of Cancer.

In The Last Decade

Abderrahim Merzak

22 papers receiving 888 citations

Peers

Abderrahim Merzak

MB

CB

ONCOL

IA

CR

Hiromasa Tsuiki Japan

Kathryn A. Grako United States

Rafaela Cañete-Soler United States

Inge Baur Germany

David R. Friedlander United States

G. J. Pilkington United Kingdom

Rupavathana Mahesparan Norway

Tomoko Matsuzaki Japan

Ulrike Ulbricht Germany

Vassiliki Kostourou United Kingdom

Hiromasa Tsuiki Japan

Abderrahim Merzak

521 ×1.0

MB

294 ×1.1

CB

202 ×0.9

ONCOL

140 ×0.7

IA

157 ×0.8

CR

Citations per year, relative to Abderrahim Merzak Abderrahim Merzak (= 1×) peers Hiromasa Tsuiki

Countries citing papers authored by Abderrahim Merzak

Since Specialization

Citations

This map shows the geographic impact of Abderrahim Merzak's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Abderrahim Merzak with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Abderrahim Merzak more than expected).

Fields of papers citing papers by Abderrahim Merzak

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Abderrahim Merzak. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Abderrahim Merzak. The network helps show where Abderrahim Merzak may publish in the future.

Co-authorship network of co-authors of Abderrahim Merzak

This figure shows the co-authorship network connecting the top 25 collaborators of Abderrahim Merzak. A scholar is included among the top collaborators of Abderrahim Merzak based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Abderrahim Merzak. Abderrahim Merzak is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

1.

Merzak, Abderrahim, et al.. (1998). Establishment and characterization of two paediatric brain tumour cell lines in vitro.. PubMed. 17(6B). 4127–34. 6 indexed citations

2.

Merzak, Abderrahim & Geoffrey J. Pilkington. (1997). Molecular and cellular pathology of intrinsic brain tumours. Cancer and Metastasis Reviews. 16(1-2). 155–177. 30 indexed citations

3.

Merzak, Abderrahim, et al.. (1996). Expression of serotonin receptors in human fetal astrocytes and glioma cell lines: a possible role in glioma cell proliferation and migration. Molecular Brain Research. 41(1-2). 1–7. 54 indexed citations

4.

Koochekpour, Shahriar, Abderrahim Merzak, & Geoffrey J. Pilkington. (1996). Vascular endothelial growth factor production is stimulated by gangliosides and TGF-β isoforms in human glioma cells in vitro. Cancer Letters. 102(1-2). 209–215. 98 indexed citations

5.

Merzak, Abderrahim, et al.. (1996). The effect of exogenous gangliosides on matrix metalloproteinase secretion by human glioma cells in vitro. European Journal of Cancer. 32(5). 868–871. 18 indexed citations

6.

Merzak, Abderrahim, Shahriar Koochekpour, & Geoffrey J. Pilkington. (1995). Adhesion of Human Glioma Cell Lines to Fibronectin, Laminin, Vitronectin and Collagen I Is Modulated by Gangliosides in vitro. Cell adhesion and communications/Cell adhesion and communication/Cell adhesion & communication. 3(1). 27–43. 39 indexed citations

7.

Koochekpour, Shahriar, Geoffrey J. Pilkington, & Abderrahim Merzak. (1995). Hyaluronic acid/CD44H interaction induces cell detachment and stimulates migration and invasion of human glioma cells in vitro. International Journal of Cancer. 63(3). 450–454. 90 indexed citations

8.

Koochekpour, Shahriar, Abderrahim Merzak, & G. J. Pilkington. (1995). Extracellular matrix proteins inhibit proliferation, upregulate migration and induce morphological changes in human glioma cell lines. European Journal of Cancer. 31(3). 375–380. 57 indexed citations

9.

Koochekpour, Shahriar, Abderrahim Merzak, & Geoffrey J. Pilkington. (1995). Growth factors and gangliosides stimulate laminin production by human glioma cells in vitro. Neuroscience Letters. 186(1). 53–56. 20 indexed citations

10.

Merzak, Abderrahim, et al.. (1995). Gangliosides modulate proliferation, migration, and invasiveness of human brain tumor cells in vitro. Molecular and Chemical Neuropathology. 24(2-3). 121–135. 33 indexed citations

11.

Koochekpour, Shahriar, et al.. (1995). VASCULAR ENDOTHELIAL GROWTH-FACTOR PRODUCTION IS STIMULATED IN RESPONSE TO GROWTH-FACTORS IN HUMAN GLIOMA-CELLS. Oncology Reports. 2(6). 1059–61. 12 indexed citations

12.

Merzak, Abderrahim, et al.. (1994). Human wild type p53 inhibits cell proliferation and elicits dramatic morphological changes in human glioma cell lines in vitro. Journal of the Neurological Sciences. 127(2). 125–133. 19 indexed citations

13.

Merzak, Abderrahim, Shahriar Koochekpour, & Geoffrey J. Pilkington. (1994). Cell surface gangliosides are involved in the control of human glioma cell invasion in vitro. Neuroscience Letters. 177(1-2). 44–46. 37 indexed citations

14.

Merzak, Abderrahim, et al.. (1994). Overexpression of the 18A2/mts1 gene and down–regulation of the TIMP–2 gene in invasive human glioma cell lines in vitro. Neuropathology and Applied Neurobiology. 20(6). 614–619. 25 indexed citations

15.

Merzak, Abderrahim, et al.. (1994). CD44 mediates human glioma cell adhesion and invasion in vitro.. PubMed. 54(15). 3988–92. 184 indexed citations

16.

Merzak, Abderrahim, et al.. (1994). Control of human glioma cell growth, migration and invasion in vitro by transforming growth factor β1. British Journal of Cancer. 70(2). 199–203. 128 indexed citations

17.

Benzakour, Omar, et al.. (1992). Transforming Growth Factor Beta Stimulates Mitogenically Mouse NIH3T3 Fibroblasts and Those Cells Transformed by the EJ-H-ras Oncogene. Growth Factors. 6(4). 265–275. 18 indexed citations

18.

Benzakour, Omar, et al.. (1992). Transforming Growth Factor Beta Stimulates Mitogenically Mouse NIH3T3 Fibroblasts and Those Cells Transformed by the EJ-H-ras Oncogene. Growth Factors. 6(3). 265–275. 8 indexed citations

19.

Merzak, Abderrahim, J. Soret, C. Martinerie, et al.. (1992). Transforming potential of truncated v-myb and stimulation of replication by gag-myb fusion products.. PubMed. 7(3). 535–42. 3 indexed citations

20.

Merzak, Abderrahim, et al.. (1992). Cooperation between the H-ras oncogene and a truncated derivative of the v-myb oncogene in transformation of hamster embryo fibroblasts.. PubMed. 7(10). 2031–9. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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