A. K. Prince

654 total citations
29 papers, 488 citations indexed

About

A. K. Prince is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Pharmacology. According to data from OpenAlex, A. K. Prince has authored 29 papers receiving a total of 488 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Cellular and Molecular Neuroscience, 10 papers in Molecular Biology and 8 papers in Pharmacology. Recurrent topics in A. K. Prince's work include Neuroscience and Neuropharmacology Research (11 papers), Cholinesterase and Neurodegenerative Diseases (8 papers) and Neurological Disorders and Treatments (5 papers). A. K. Prince is often cited by papers focused on Neuroscience and Neuropharmacology Research (11 papers), Cholinesterase and Neurodegenerative Diseases (8 papers) and Neurological Disorders and Treatments (5 papers). A. K. Prince collaborates with scholars based in United Kingdom, United States and Canada. A. K. Prince's co-authors include C.K. Atterwill, Raj N. Kalaria, K S Ishimoto, Stephen Lory, Lisa Saiman, Christopher Hudson, Joe D. Pagan, Patricia A. Harris, Raymond J. Geor and Ann E. Kingsbury and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Brain Research and FEBS Letters.

In The Last Decade

A. K. Prince

25 papers receiving 453 citations

Peers

A. K. Prince

CMN

PHARM

EQUIN

Allan White Chile

JUDITH M. HUNT United Kingdom

C. Girardi Italy

Tetsuro Taneike Japan

Haolin Zhang China

Anthony M. Gawienowski United States

Angela Catapano Italy

M. Cirino Canada

Takenori Onaga Japan

Gregory W. Sawyer United States

Allan White Chile

A. K. Prince

208 ×1.0

76 ×0.6

CMN

36 ×0.4

PHARM

126 ×1.9

103 ×1.7

EQUIN

Citations per year, relative to A. K. Prince A. K. Prince (= 1×) peers Allan White

Countries citing papers authored by A. K. Prince

Since Specialization

Citations

This map shows the geographic impact of A. K. Prince's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. K. Prince with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. K. Prince more than expected).

Fields of papers citing papers by A. K. Prince

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. K. Prince. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. K. Prince. The network helps show where A. K. Prince may publish in the future.

Co-authorship network of co-authors of A. K. Prince

This figure shows the co-authorship network connecting the top 25 collaborators of A. K. Prince. A scholar is included among the top collaborators of A. K. Prince based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A. K. Prince. A. K. Prince is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Prince, A. K., Hanna Rennert, Danielle Brandman, et al.. (2025). Common bile duct dilatation on MRI in autosomal dominant polycystic kidney disease. Abdominal Radiology. 51(2). 758–767.

Prince, A. K., et al.. (2002). Comparison of the metabolic responses of trained Arabians and Thoroughbreds during high‐ and low‐intensity exercise. Equine Veterinary Journal. 34(S34). 95–99. 50 indexed citations

Pagan, Joe D., et al.. (2002). Effects of fat adaptation on glucose kinetics and substrate oxidation during low‐intensity exercise. Equine Veterinary Journal. 34(S34). 33–38. 33 indexed citations

Jones, H. B., et al.. (1993). Effects of the neurotoxin ethylcholine mustard aziridinium in rat brain reaggregate cultures. Toxicology in Vitro. 7(1). 69–80. 1 indexed citations

Jones, H. B., et al.. (1993). Morphological assessment of ethyl choline mustard aziridinium-induced neurotoxicity in rat brain reaggregate cultures. Acta Neuropathologica. 86(2). 154–162. 1 indexed citations

Saiman, Lisa, K S Ishimoto, Stephen Lory, & A. K. Prince. (1990). The Effect of Piliation and Exoproduct Expression on the Adherence of Pseudomonas aeruginosa to Respiratory Epithelial Monolayers. The Journal of Infectious Diseases. 161(3). 541–548. 98 indexed citations

Atterwill, C.K., et al.. (1989). Effect of nerve growth factor and thyrotropin releasing hormone on cholinergic neurones in developing rat brain reaggregate cultures lesioned with ethylcholine mustard aziridinium. Biochemical Pharmacology. 38(10). 1631–1638. 5 indexed citations

Prince, A. K., et al.. (1988). The neurotoxicity of ethylcholine mustard aziridinium (ECMA) in rat brain reaggregate cultures. Toxicology. 49(1). 115–119. 9 indexed citations

Kalaria, Rajesh N. & A. K. Prince. (1988). Neurochemical development of the striatum in a precocial (guinea pig) and an altricial (rat) species. International Journal of Developmental Neuroscience. 6(2). 161–166. 4 indexed citations

10.

Prince, A. K., et al.. (1987). High-affinity uptake of choline, a marker for cholinergic nerve terminals, is not specific in developing rat brain. Developmental Brain Research. 35(2). 175–181. 27 indexed citations

11.

Prince, A. K., et al.. (1987). The Effect of the Cholinergic Neurotoxin ECMA on Neural Function in Brain Reaggregate Cultures. PubMed. 11. 243–246. 3 indexed citations

12.

Norman, Andrew B., et al.. (1987). Supersensitivity of d-amphetamine-induced hyperthermia in rats following continuous treatment with neuroleptics. European Journal of Pharmacology. 140(3). 349–351. 2 indexed citations

13.

Kalaria, Raj N. & A. K. Prince. (1985). Effects of thyroid deficiency on the development of cholinergic, GABA, dopaminergic and glutamate neuron markers and DNA concentrations in the rat corpus striatum. International Journal of Developmental Neuroscience. 3(6). 655–666. 36 indexed citations

14.

Atterwill, C.K., Ann E. Kingsbury, Julia Nicholls, & A. K. Prince. (1984). Development of markers for cholinergic neurones in re‐aggregate cultures of foetal rat whole brain in serum‐containing and serum‐free media: effects of triiodothyronine (T₃). British Journal of Pharmacology. 83(1). 89–102. 38 indexed citations

15.

Prince, A. K.. (1978). Chemical pharmacology of the synapse. FEBS Letters. 89(2). 348–349.

16.

Atterwill, C.K., A. K. Prince, & G.G. Brownlee. (1978). Choline uptake, choline acetyltransferase and the acetylation of choline in chick and rat brain [proceedings].. PubMed. 62(3). 398P–398P.

17.

Prince, A. K., et al.. (1973). A stain for the detection of choline acetyltransferase.. PubMed. 47(3). 670P–671P.

18.

Prince, A. K.. (1967). Properties of choline acetyltransferase isolated from squid ganglia.. Proceedings of the National Academy of Sciences. 57(4). 1117–1122. 18 indexed citations

19.

Prince, A. K.. (1966). A sensitive fluorometric procedure for the determination of small quantities of acetylcholinesterase. Biochemical Pharmacology. 15(4). 411–417. 22 indexed citations

20.

Prince, A. K.. (1966). Spectrophotometric study of the acetylcholinesterase-catalyzed hydrolysis of 1-methyl-acetoxyquinolinium iodides. Archives of Biochemistry and Biophysics. 113(1). 195–204. 34 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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