A. Calcagnile

1.7k total citations
30 papers, 1.2k citations indexed

About

A. Calcagnile is a scholar working on Molecular Biology, Cancer Research and Epidemiology. According to data from OpenAlex, A. Calcagnile has authored 30 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 11 papers in Cancer Research and 4 papers in Epidemiology. Recurrent topics in A. Calcagnile's work include DNA Repair Mechanisms (13 papers), Carcinogens and Genotoxicity Assessment (10 papers) and CRISPR and Genetic Engineering (6 papers). A. Calcagnile is often cited by papers focused on DNA Repair Mechanisms (13 papers), Carcinogens and Genotoxicity Assessment (10 papers) and CRISPR and Genetic Engineering (6 papers). A. Calcagnile collaborates with scholars based in Italy, United States and Netherlands. A. Calcagnile's co-authors include Eugenia Dogliotti, Mariarosaria D’Errico, Miria Stefanini, Paolo Degan, Eleonora Parlanti, Giovanna Zambruno, Massimo Teson, Francesco Sera, Alessandro Giuliani and Fabio Palombo and has published in prestigious journals such as Nucleic Acids Research, The EMBO Journal and Cancer Research.

In The Last Decade

A. Calcagnile

29 papers receiving 1.2k citations

Peers

A. Calcagnile

ONCOL

DERMA

GENET

Mariarosaria D’Errico Italy

Ye Jin Jang South Korea

Daniel Chang United States

Eileen M. Rogan Australia

H U Kontny United States

Sahn-Ho Kim United States

R P Huang United States

Nobuyasu Komazawa Japan

Alfred May United States

Lidia Avalle Italy

Mariarosaria D’Errico Italy

A. Calcagnile

1.3k ×1.3

431 ×1.1

340 ×1.5

ONCOL

290 ×2.2

DERMA

187 ×1.5

GENET

Citations per year, relative to A. Calcagnile A. Calcagnile (= 1×) peers Mariarosaria D’Errico

Countries citing papers authored by A. Calcagnile

Since Specialization

Citations

This map shows the geographic impact of A. Calcagnile's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. Calcagnile with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. Calcagnile more than expected).

Fields of papers citing papers by A. Calcagnile

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. Calcagnile. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. Calcagnile. The network helps show where A. Calcagnile may publish in the future.

Co-authorship network of co-authors of A. Calcagnile

This figure shows the co-authorship network connecting the top 25 collaborators of A. Calcagnile. A scholar is included among the top collaborators of A. Calcagnile based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A. Calcagnile. A. Calcagnile is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Medda, Emanuela, Anna Minoprio, Lorenza Nisticò, et al.. (2016). The response to oxidative stress and metallomics analysis in a twin study: The role of the environment. Free Radical Biology and Medicine. 97. 236–243. 4 indexed citations

Parlanti, Eleonora, Mariarosaria D’Errico, Paolo Degan, et al.. (2012). The cross talk between pathways in the repair of 8-oxo-7,8-dihydroguanine in mouse and human cells. Free Radical Biology and Medicine. 53(11). 2171–2177. 44 indexed citations

Pascucci, Barbara, Egidio Iorio, Sara Giovannini, et al.. (2012). An altered redox balance mediates the hypersensitivity of Cockayne syndrome primary fibroblasts to oxidative stress. Aging Cell. 11(3). 520–529. 83 indexed citations

D’Errico, Mariarosaria, Emanuele de Rinaldis, Monica Francesca Blasi, et al.. (2008). Genome-wide expression profile of sporadic gastric cancers with microsatellite instability. European Journal of Cancer. 45(3). 461–469. 262 indexed citations

D’Errico, Mariarosaria, et al.. (2006). Cell type and DNA damage specific response of human skin cells to environmental agents. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 614(1-2). 37–47. 83 indexed citations

D’Errico, Mariarosaria, Eleonora Parlanti, Massimo Teson, et al.. (2006). New functions of XPC in the protection of human skin cells from oxidative damage. The EMBO Journal. 25(18). 4305–4315. 210 indexed citations

D’Errico, Mariarosaria, Massimo Teson, A. Calcagnile, et al.. (2004). Characterization of the ultraviolet B and X-ray response of primary cultured epidermal cells from patients with disseminated superficial actinic porokeratosis. British Journal of Dermatology. 150(1). 47–55. 7 indexed citations

D’Errico, Mariarosaria, A. Calcagnile, Biagio Didona, et al.. (2000). UV mutation signature in tumor suppressor genes involved in skin carcinogenesis in xeroderma pigmentosum patients. Oncogene. 19(3). 463–467. 50 indexed citations

D’Errico, Mariarosaria, A. Calcagnile, Ivano Iavarone, et al.. (1999). Factors that influence the DNA repair capacity of normal and skin cancer-affected individuals.. PubMed. 8(6). 553–9. 45 indexed citations

10.

D’Errico, Mariarosaria, A. Calcagnile, Rosamaria Corona, et al.. (1997). p53 mutations and chromosome instability in basal cell carcinomas developed at an early or late age.. PubMed. 57(4). 747–52. 59 indexed citations

11.

Calcagnile, A.. (1996). Misincorporation rate and type on the leading and lagging strands of UV- damaged DNA. Nucleic Acids Research. 24(15). 3005–3009. 10 indexed citations

12.

Meschini, Roberta, et al.. (1995). Strand bias of ultraviolet light‐induced mutations in a transcriptionally active gene in human cells. Molecular Carcinogenesis. 14(3). 214–225. 11 indexed citations

13.

Fortini, Paola, A. Calcagnile, Alfonso Di Muccio, Margherita Bignami, & Eugenia Dogliotti. (1993). Quantitative relationship between ethylated DNA bases and gene mutation at two loci in CHO cells. Environmental and Molecular Mutagenesis. 21(2). 154–159. 11 indexed citations

14.

Fortini, Paola, et al.. (1993). Mutagenic processing of ethylation damage in mammalian cells: the use of methoxyamine to study apurinic/apyrimidinic site-induced mutagenesis.. PubMed. 53(5). 1149–55. 26 indexed citations

15.

Fortini, Paola, et al.. (1992). Methoxyamine modification of abasic sites protects CHO cells from the cytotoxic and mutagenic effects of oxygen alkylation. Carcinogenesis. 13(1). 87–93. 17 indexed citations

16.

Benigni, Romualdo, A. Calcagnile, Alessandro Giuliani, & P. Leopardi. (1990). Inhibition of replicative DNA synthesis and induction of DNA repair in human fibroblasts by the intercalating drugs proflavine and 9‐aminoacridine. Journal of Toxicology and Environmental Health. 31(2). 117–124. 1 indexed citations

17.

Bignami, Margherita, Alfonso Di Muccio, A. Calcagnile, et al.. (1988). Relationship between specific alkylated bases and mutations at two gene loci induced by ethylnitrosourea and diethyl sulfate in CHO cells. Mutation Research/DNA Repair Reports. 193(1). 43–51. 14 indexed citations

18.

Zijno, Andrea, et al.. (1987). Cytotoxicity, mutations and SCEs induced by methylating agents are reduced in CHO cells expressing an active mammalian O⁶-methylguanine-DNA methyltransferase gene. Carcinogenesis. 8(10). 1417–1421. 16 indexed citations

19.

Benigni, Romualdo, et al.. (1984). Biological monitoring of workers in the rubber industry. Mutation Research/Genetic Toxicology. 138(1). 105–111. 12 indexed citations

20.

Benigni, Romualdo, Eugenia Dogliotti, E. Falcone, & A. Calcagnile. (1982). DNA repair studies on diallate, triallate and sulfallate in human cell cultures. Mutation Research Letters. 103(3-6). 385–390. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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