Wuting Wei

Curcumin plays neuroprotective roles against traumatic brain injury partly via Nrf2 signaling

NRF2 and NF-қB interplay in cerebrovascular and neurodegenerative disorders: Molecular mechanisms and possible therapeutic approaches

Role of Nrf2 and protective effects of Metformin against tobacco smoke-induced cerebrovascular toxicity

Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice

Defining roles of specific reactive oxygen species (ROS) in cell biology and physiology

The Flavone Luteolin Improves Central Nervous System Disorders by Different Mechanisms: A Review

Curcumin provides neuroprotection in model of traumatic brain injury via the Nrf2-ARE signaling pathway

Therapeutic targeting of the NRF2 and KEAP1 partnership in chronic diseases

<p>Reactive Oxygen Species: Drivers of Physiological and Pathological Processes</p>

Fisetin alleviates oxidative stress after traumatic brain injury via the Nrf2-ARE pathway

Altered Nrf2 Signaling Mediates Hypoglycemia-Induced Blood–Brain Barrier Endothelial Dysfunction In Vitro

Autophagy in acute brain injury

Luteolin as an anti-inflammatory and neuroprotective agent: A brief review

Inhibition of the NRF2/KEAP1 Axis: A Promising Therapeutic Strategy to Alter Redox Balance of Cancer Cells

Induction of the Vitamin D Receptor Attenuates Autophagy Dysfunction-Mediated Cell Death Following Traumatic Brain Injury.

Important Flavonoids and Their Role as a Therapeutic Agent

Genetic elimination of Nrf2 aggravates secondary complications except for vasospasm after experimental subarachnoid hemorrhage in mice

Posttraumatic administration of luteolin protects mice from traumatic brain injury: Implication of autophagy and inflammation

Nrf2-ARE signaling provides neuroprotection in traumatic brain injury via modulation of the ubiquitin proteasome system

Luteolin provides neuroprotection in models of traumatic brain injury via the Nrf2–ARE pathway