Seiko Ohno

2014 AHA/ACC/HRS Guideline for the Management of Patients With Atrial Fibrillation: Executive Summary

Exonic mutations in SCN9A (Na _V 1.7) are found in a minority of patients with erythromelalgia

Long QT Syndrome: Genetics and Future Perspective

2016 MASCC and ESMO guideline update for the prevention of chemotherapy- and radiotherapy-induced nausea and vomiting and of nausea and vomiting in advanced cancer patients

Molecular characterization of two founder mutations causing long QT syndrome and identification of compound heterozygous patients

Voltage-Gated Sodium Channels: Mutations, Channelopathies and Targets

Cell alignment induced by anisotropic electrospun fibrous scaffolds alone has limited effect on cardiomyocyte maturation

Applications of genome editing technology in the targeted therapy of human diseases: mechanisms, advances and prospects

Reference glycan structure libraries of primary human cardiomyocytes and pluripotent stem cell-derived cardiomyocytes reveal cell-type and culture stage-specific glycan phenotypes

Risk of Fatal Arrhythmic Events in Long QT Syndrome Patients After Syncope

The changing landscape of atherosclerosis

The genetic basis of long QT and short QT syndromes: A mutation update

ACC/AHA/HRS 2008 Guidelines for Device-Based Therapy of Cardiac Rhythm Abnormalities

Modeling the human Na_v1.5 sodium channel: structural and mechanistic insights of ion permeation and drug blockade

5-Hydroxytryptamine₃receptor antagonists and cardiac side effects

A structure-based computational workflow to predict liability and binding modes of small molecules to hERG

Recent advances in the molecular pathophysiology of atrial fibrillation

De Novo Mutation in the SCN5A Gene Associated with Brugada Syndrome

Heart mitochondria: gates of life and death

Risk Stratification in the Long-QT Syndrome

A Pragmatic Guide to Enrichment Strategies for Mass Spectrometry–Based Glycoproteomics

Cardiac Channel Molecular Autopsy: Insights From 173 Consecutive Cases of Autopsy-Negative Sudden Unexplained Death Referred for Postmortem Genetic Testing

Cardiac sodium channelopathy associated with SCN5A mutations: electrophysiological, molecular and genetic aspects

Ranolazine inhibition of hERG potassium channels: Drug–pore interactions and reduced potency against inactivation mutants

The usual suspects in sudden cardiac death of the young: a focus on inherited arrhythmogenic diseases

Engineering Adolescence

2014 AHA/ACC/HRS Guideline for the Management of Patients With Atrial Fibrillation

Electrospinning and Electrospun Nanofibers: Methods, Materials, and Applications

Crystallographic insights into sodium-channel modulation by the β4 subunit

Control of inositol 1,4,5-trisphosphate-induced Ca2+ release by cytosolic Ca2+

The Role of Voltage-Gated Sodium Channels in Pain Signaling

Genetics of Atrial Fibrillation

2015 ESC Guidelines for the management of patients with ventricular arrhythmias and the prevention of sudden cardiac death

Model for long QT syndrome type 2 using human iPS cells demonstrates arrhythmogenic characteristics in cell culture

Neuropathic Pain: From Mechanisms to Treatment

ACC/AHA/HRS 2008 Guidelines for Device-Based Therapy of Cardiac Rhythm Abnormalities

Human Induced Pluripotent Stem Cell–Derived Cardiomyocytes

2014 AHA/ACC/HRS Guideline for the Management of Patients With Atrial Fibrillation

Pharmacology of the Na _v 1.1 domain IV voltage sensor reveals coupling between inactivation gating processes

Role of implantable cardioverter defibrillator therapy in patients with long QT syndrome

Andersen–Tawil syndrome: Clinical and molecular aspects

Genetic Characteristics of Children and Adolescents With Long-QT Syndrome Diagnosed by School-Based Electrocardiographic Screening Programs

Hypermorphic mutation of the voltage-gated sodium channel encoding gene Scn10a causes a dramatic stimulus-dependent neurobehavioral phenotype

NovelSCN10Avariants associated with Brugada syndrome

OJ-347 Noninducibility of Atrial Fibrillation as an End Point of Pulmonary Vein Antrum Isolation for Paroxysmal Atrial Fibrillation(Arrhythmia, therapy-5 (A) OJ58,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)

D85N, a KCNE1 Polymorphism, Is a Disease-Causing Gene Variant in Long QT Syndrome

A Novel SCN5A Gain-of-Function Mutation M1875T Associated With Familial Atrial Fibrillation

Mechanistic basis for the pathogenesis of long QT syndrome associated with a common splicing mutation in KCNQ1 gene

N- and C-terminal KCNE1 mutations cause distinct phenotypes of long QT syndrome

Complex aberrant splicing in the induced pluripotent stem cell–derived cardiomyocytes from a patient with long QT syndrome carrying KCNQ1-A344Aspl mutation

A novel gain-of-function KCNJ2 mutation associated with short-QT syndrome impairs inward rectification of Kir2.1 currents

Additional Gene Variants Reduce Effectiveness of Beta‐Blockers in the LQT1 Form of Long QT Syndrome

A KCNQ1 mutation contributes to the concealed type 1 long QT phenotype by limiting the Kv7.1 channel conformational changes associated with protein kinase A phosphorylation

Long QT syndrome with compound mutations is associated with a more severe phenotype: A Japanese multicenter study

Age‐ and Genotype‐Specific Triggers for Life‐Threatening Arrhythmia in the Genotyped Long QT Syndrome

Mutation site-specific differences in arrhythmic risk and sensitivity to sympathetic stimulation in the LQT1 form of congenital long QT syndrome

Ultrastructural Maturation of Human-Induced Pluripotent Stem Cell-Derived Cardiomyocytes in a Long-Term Culture

Mutation Analysis of the Glycerol-3 Phosphate Dehydrogenase-1 Like (GPD1L) Gene in Japanese Patients With Brugada Syndrome

A Novel KCNJ2 Nonsense Mutation, S369X, Impedes Trafficking and Causes a Limited Form of Andersen-Tawil Syndrome

Phenotype Variability in Patients CarryingKCNJ2Mutations

Novel <i>SCN3B</i> Mutation Associated With Brugada Syndrome Affects Intracellular Trafficking and Function of Nav1.5

Pharmacological correction of long QT-linked mutations in KCNH2 (hERG) increases the trafficking of Kv11.1 channels stored in the transitional endoplasmic reticulum