Scott J. Koppel

Diminished O-GlcNAcylation in Alzheimer's disease is strongly correlated with mitochondrial anomalies

Reactive oxygen species (ROS) as pleiotropic physiological signalling agents

O-GlcNAc modification of small heat shock proteins enhances their anti-amyloid chaperone activity

Bioenergetics and translational metabolism: implications for genetics, physiology and precision medicine

Functional crosstalk among oxidative stress and O-GlcNAc signaling pathways

Sustained O-GlcNAcylation reprograms mitochondrial function to regulate energy metabolism

Platelet cytochrome oxidase and citrate synthase activities in APOE ε4 carrier and non-carrier Alzheimer's disease patients