Sandra Baches

Current knowledge on exosome biogenesis and release

Apolipoprotein E Regulates Amyloid Formation within Endosomes of Pigment Cells

Interactions between the microbiota, immune and nervous systems in health and disease

The glymphatic pathway in neurological disorders

Sphingolipids and their metabolism in physiology and disease

The Tetraspanin CD63 Regulates ESCRT-Independent and -Dependent Endosomal Sorting during Melanogenesis

Principles of nanoparticle design for overcoming biological barriers to drug delivery

Neutral Sphingomyelinase-2 Deficiency Ameliorates Alzheimer's Disease Pathology and Improves Cognition in the 5XFAD Mouse

Mutations in or near the Transmembrane Domain Alter PMEL Amyloid Formation from Functional to Pathogenic

Understanding the functions and relationships of the glymphatic system and meningeal lymphatics

Blood–brain barrier breakdown in Alzheimer disease and other neurodegenerative disorders

Quantitative profiling of the protein coronas that form around nanoparticles

Gut Microbiota Regulate Motor Deficits and Neuroinflammation in a Model of Parkinson’s Disease

Alzheimer Disease: An Update on Pathobiology and Treatment Strategies

Shedding light on the cell biology of extracellular vesicles

Alzheimer’s Disease-Associated β-Amyloid Is Rapidly Seeded by Herpesviridae to Protect against Brain Infection

The role of short-chain fatty acids in microbiota–gut–brain communication

The Amyloid State of Proteins in Human Diseases

Reassessment of Exosome Composition

Neuroinflammation and microglial activation in Alzheimer disease: where do we go from here?

Extracellular vesicles: Exosomes, microvesicles, and friends

Overview of Extracellular Vesicles, Their Origin, Composition, Purpose, and Methods for Exosome Isolation and Analysis

A cellular complex of BACE1 and γ-secretase sequentially generates Aβ from its full-length precursor

Amyloid-β peptide protects against microbial infection in mouse and worm models of Alzheimer’s disease

Single-nanometer iron oxide nanoparticles as tissue-permeable MRI contrast agents

How neuroinflammation contributes to neurodegeneration

Insulin differentially affects the distribution kinetics of amyloid beta 40 and 42 in plasma and brain

The cholinergic system in the pathophysiology and treatment of Alzheimer’s disease

Immunolesion‐induced loss of cholinergic projection neurones promotes β‐amyloidosis and tau hyperphosphorylation in the hippocampus of triple‐transgenic mice

Biogenesis, Secretion, and Intercellular Interactions of Exosomes and Other Extracellular Vesicles

Formation of Pmel17 Amyloid Is Regulated by Juxtamembrane Metalloproteinase Cleavage, and the Resulting C-terminal Fragment Is a Substrate for γ-Secretase

Nanoparticulate flurbiprofen reduces amyloid-β42 generation in an in vitro blood–brain barrier model

Presenilin Is the Molecular Target of Acidic γ-Secretase Modulators in Living Cells

Presenilin-1 but not amyloid precursor protein mutations present in mouse models of Alzheimer’s disease attenuate the response of cultured cells to γ-secretase modulators regardless of their potency and structure

Endothelial LRP1 transports amyloid-β1–42 across the blood-brain barrier