Ruby Mishra

Cellular and Molecular Mechanisms Associated with Carotid Body Adaptations to Chronic Hypoxia

Hypoxia-inducible factor 2α (HIF-2α) heterozygous-null mice exhibit exaggerated carotid body sensitivity to hypoxia, breathing instability, and hypertension

Neural regulation of hypoxia-inducible factors and redox state drives the pathogenesis of hypertension in a rodent model of sleep apnea

Induction of HIF‐1α expression by intermittent hypoxia: Involvement of NADPH oxidase, Ca²⁺ signaling, prolyl hydroxylases, and mTOR

Heterozygous HIF‐1α deficiency impairs carotid body‐mediated systemic responses and reactive oxygen species generation in mice exposed to intermittent hypoxia

Transcriptional responses to intermittent hypoxia

Ca2+/Calmodulin Kinase-dependent Activation of Hypoxia Inducible Factor 1 Transcriptional Activity in Cells Subjected to Intermittent Hypoxia

HIF-1 Inhibits Mitochondrial Biogenesis and Cellular Respiration in VHL-Deficient Renal Cell Carcinoma by Repression of C-MYC Activity

Role of c-fos in hypoxia-induced AP-1 cis-element activity and tyrosine hydroxylase gene expression

Role of oxidative stress in intermittent hypoxia‐induced immediate early gene activation in rat PC12 cells

Oxygen sensing in the body

HIF-1α Activation by Intermittent Hypoxia Requires NADPH Oxidase Stimulation by Xanthine Oxidase

Carotid body hyperplasia and enhanced ventilatory responses to hypoxia in mice with heterozygous deficiency of PHD2

HIF-1–Dependent Respiratory, Cardiovascular, and Redox Responses to Chronic Intermittent Hypoxia

ROS Signaling in Systemic and Cellular Responses to Chronic Intermittent Hypoxia

Adaptive and Maladaptive Cardiorespiratory Responses to Continuous and Intermittent Hypoxia Mediated by Hypoxia-Inducible Factors 1 and 2

L-type Ca²⁺channel activation regulates induction of c-fostranscription by hypoxia

Regulation of Gene Expression by HIF‐1

Pathophysiology of Sleep Apnea

The role of hypoxia‐inducible factors in carotid body (patho) physiology

Regulation of Oxygen Homeostasis by Hypoxia-Inducible Factor 1

Induction of immediate early response genes by hypoxia. Possible molecular bases for systems adaptation to low pO2.