Pan Chang

Panax Notoginseng Saponins Protect Cardiac Myocytes Against Endoplasmic Reticulum Stress and Associated Apoptosis Through Mediation of Intracellular Calcium Homeostasis

Reactive oxygen species, toxicity, oxidative stress, and antioxidants: chronic diseases and aging

Ferroptosis: molecular mechanisms and health implications

Ferroptosis: Death by Lipid Peroxidation

Macrolides sensitize EGFR-TKI-induced non-apoptotic cell death via blocking autophagy flux in pancreatic cancer cell lines

Chemical constituents of Panax ginseng and Panax notoginseng explain why they differ in therapeutic efficacy

Autophagy in acute brain injury

Modes of Physiologic H ₂ S Signaling in the Brain and Peripheral Tissues

Programmed necrosis and necroptosis signalling

Necroptosis in health and diseases

Protective effects of ex‐527 on cerebral ischemia–reperfusion injury through necroptosis signaling pathway attenuation

Neuronal Death After Hemorrhagic Stroke In Vitro and In Vivo Shares Features of Ferroptosis and Necroptosis

Induction of the Vitamin D Receptor Attenuates Autophagy Dysfunction-Mediated Cell Death Following Traumatic Brain Injury.

Protein kinase G–regulated production of H ₂ S governs oxygen sensing

Caspase‐8: regulating life and death

Necrostatin-1 Suppresses Autophagy and Apoptosis in Mice Traumatic Brain Injury Model

Hydrogen Sulfide Offers Neuroprotection on Traumatic Brain Injury in Parallel with Reduced Apoptosis and Autophagy in Mice

B-type natriuretic peptide attenuates endoplasmic reticulum stress in H9c2 cardiomyocytes underwent hypoxia/reoxygenation injury under high glucose/high fat conditions

Anti-Necroptosis Chemical Necrostatin-1 Can Also Suppress Apoptotic and Autophagic Pathway to Exert Neuroprotective Effect in Mice Intracerebral Hemorrhage Model