Mario Hebecker

Modulation of immune cell reactivity with cis -binding Siglec agonists

Treml4, an Ig Superfamily Member, Mediates Presentation of Several Antigens to T Cells In Vivo, Including Protective Immunity to HER2 Protein

Engineering precision nanoparticles for drug delivery

Discovery of serum proteomic biomarkers for prediction of response to infliximab (a monoclonal anti-TNF antibody) treatment in rheumatoid arthritis: An exploratory analysis

Messenger RNA-Based Vaccines Against Infectious Diseases

Rheumatoid arthritis

Complement, a target for therapy in inflammatory and degenerative diseases

Role of C-Reactive Protein at Sites of Inflammation and Infection

Anti-complement-factor H-associated glomerulopathies

Bypassing adverse injection reactions to nanoparticles through shape modification and attachment to erythrocytes

Be on Target: Strategies of Targeting Alternative and Lectin Pathway Components in Complement-Mediated Diseases

Complement associated microvascular injury and thrombosis in the pathogenesis of severe COVID-19 infection: A report of five cases

Inflammation and tumor progression: signaling pathways and targeted intervention

Molecular mechanisms of necroptosis: an ordered cellular explosion

The immunopathology of sepsis and potential therapeutic targets

Molecular mechanisms of late apoptotic/necrotic cell clearance

Guidelines on the Use of Therapeutic Apheresis in Clinical Practice – Evidence‐Based Approach from the Writing Committee of the American Society for Apheresis: The Eighth Special Issue

Diversity, Mechanisms, and Significance of Macrophage Plasticity

Klebsiella pneumoniae: Going on the Offense with a Strong Defense

Complement System Part II: Role in Immunity

PTX3, a Humoral Pattern Recognition Molecule, in Innate Immunity, Tissue Repair, and Cancer

PEGylated liposomes: immunological responses

Modulation of three key innate immune pathways for the most common retinal degenerative diseases

Complement in removal of the dead – balancing inflammation

Monomeric C‐reactive protein modulates classic complement activation on necrotic cells

Molecular basis of C-reactive protein binding and modulation of complement activation by factor H-related protein 4

Selectivity of C3-opsonin targeted complement inhibitors: A distinct advantage in the protection of erythrocytes from paroxysmal nocturnal hemoglobinuria patients

Human complement factor H-related protein 4 binds and recruits native pentameric C-reactive protein to necrotic cells

Factor H inhibits complement activation induced by liposomal and micellar drugs and the therapeutic antibody rituximab in vitro

Factor H-related Protein 4 Activates Complement by Serving as a Platform for the Assembly of Alternative Pathway C3 Convertase via Its Interaction with C3b Protein

Factor H: A Complement Regulator in Health and Disease, and a Mediator of Cellular Interactions

An Engineered Construct Combining Complement Regulatory and Surface-Recognition Domains Represents a Minimal-Size Functional Factor H

Factor H–Related Protein 5 Interacts with Pentraxin 3 and the Extracellular Matrix and Modulates Complement Activation