Standout Papers

Glutathione S-Transferases 1974 2026 1991 2008 16.0k
  1. Glutathione S-Transferases (1974)
    William H. Habig, M. Pabst et al. Journal of Biological Chemistry

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Works of M. Pabst being referenced

Removal of endotoxin from protein solutions by phase separation using triton X-114
1990
Lipopolysaccharide priming of human neutrophils for an enhanced respiratory burst. Role of intracellular free calcium.
1989
Invasive M1T1 group A Streptococcus undergoes a phase‐shift in vivo to prevent proteolytic degradation of multiple virulence factors by SpeB
2003
Attachment of Fusobacterium nucleatum to Fibronectin Immobilized on Gingival Epithelial Cells or Glass Coverslips
1995
Enhancement by Ca2+ or Mg2+ of catalytic activity of the superoxide-producing NADPH oxidase in membrane fractions of human neutrophils and monocytes.
1985
Mercapturic acid formation: The several glutathione transferases of rat liver
1973
Activation of macrophages for enhanced release of superoxide anion and greater killing of Candida albicans by injection of muramyl dipeptide.
1980
Glutathione S-Transferases
1974 Standout
Increased production of superoxide anion by macrophages exposed in vitro to muramyl dipeptide or lipopolysaccharide.
1980
Chronic infection due to Mycobacterium intracellulare in mice: association with macrophage release of prostaglandin E2 and reversal by injection of indomethacin, muramyl dipeptide, or interferon-gamma.
1986
Inhibition of macrophage priming by sulfatide from Mycobacterium tuberculosis.
1988
CD18-deficient cells respond to lipopolysaccharide in vitro.
1990
Priming of neutrophils and macrophages for enhanced release of superoxide anion by the calcium ionophore ionomycin. Implications for regulation of the respiratory burst.
1987
The Identity of Glutathione S -Transferase B with Ligandin, a Major Binding Protein of Liver
1974
Cultured human monocytes require exposure to bacterial products to maintain an optimal oxygen radical response.
1982
Monocyte responses to sulfatide from Mycobacterium tuberculosis: inhibition of priming for enhanced release of superoxide, associated with increased secretion of interleukin-1 and tumor necrosis factor alpha, and altered protein phosphorylation
1991
Rankless by CCL
2026