Luoluo Wang

Activation of the p62‐Keap1‐NRF2 pathway protects against ferroptosis in hepatocellular carcinoma cells

Tumour initiating cells and IGF/FGF signalling contribute to sorafenib resistance in hepatocellular carcinoma

Ferroptosis: past, present and future

The mechanisms of sorafenib resistance in hepatocellular carcinoma: theoretical basis and therapeutic aspects

Ferroptosis: molecular mechanisms and health implications

Hepatocellular carcinoma

The molecular machinery of regulated cell death

A central hydrophobic E1 region controls the pH range of hepatitis C virus membrane fusion and susceptibility to fusion inhibitors

Inflammation and tumor progression: signaling pathways and targeted intervention

Natural product-based nanoformulations for cancer therapy: Opportunities and challenges

Broadening horizons: the role of ferroptosis in cancer

Targeting ferroptosis as a vulnerability in cancer

Mechanisms of resistance to sorafenib and the corresponding strategies in hepatocellular carcinoma

A polymeric nanoparticle formulation of curcumin in combination with sorafenib synergistically inhibits tumor growth and metastasis in an orthotopic model of human hepatocellular carcinoma

Sigma‐1 receptor protects against ferroptosis in hepatocellular carcinoma cells

Turmeric (Curcuma longa) and its major constituent (curcumin) as nontoxic and safe substances: Review

The oral bioavailability of curcumin from micronized powder and liquid micelles is significantly increased in healthy humans and differs between sexes

Metallothionein‐1G facilitates sorafenib resistance through inhibition of ferroptosis

Autophagy promotes ferroptosis by degradation of ferritin

Turmeric curcumin inhibits entry of all hepatitis C virus genotypes into human liver cells

Hypoxia-mediated sorafenib resistance can be overcome by EF24 through Von Hippel-Lindau tumor suppressor-dependent HIF-1α inhibition in hepatocellular carcinoma

In Vivo and In Vitro Suppression of Hepatocellular Carcinoma by EF24, a Curcumin Analog