Kyun‐Do Kim

Regulatory T cells in autoimmune kidney diseases and transplantation

Ferroptosis: molecular mechanisms and health implications

Transcriptional and epigenetic basis of Treg cell development and function: its genetic anomalies or variations in autoimmune diseases

Accurate de novo design of heterochiral protein–protein interactions

Mechanisms, regulation and functions of the unfolded protein response

Combined Immunodeficiencies with Nonfunctional T Lymphocytes

The role of iron and reactive oxygen species in cell death

Reactive oxygen species (ROS) as pleiotropic physiological signalling agents

CRAC Channels and Calcium Signaling in T Cell-Mediated Immunity

Intracellular Ca2+ Sensing: Its Role in Calcium Homeostasis and Signaling

The plasma membrane channel ORAI1 mediates detrimental calcium influx caused by endogenous oxidative stress

Photothermal-Chemotherapy Enhancing Tumor Immunotherapy by Multifunctional Metal–Organic Framework Based Drug Delivery System

Regulatory T Cells and Human Disease

Here, there, and everywhere: The importance of ER membrane contact sites

Ion Channels in Innate and Adaptive Immunity

Piezo1 channels restrain regulatory T cells but are dispensable for effector CD4 ⁺ T cell responses

Photothermal Nanomaterials: A Powerful Light-to-Heat Converter

Selective ORAI1 Inhibition Ameliorates Autoimmune Central Nervous System Inflammation by Suppressing Effector but Not Regulatory T Cell Function

Junctate is a Ca ^{2 +} -sensing structural component of Orai1 and stromal interaction molecule 1 (STIM1)

A novel EF-hand protein, CRACR2A, is a cytosolic Ca2+ sensor that stabilizes CRAC channels in T cells

Targeted calcium influx boosts cytotoxic T lymphocyte function in the tumour microenvironment

ORAI1 Deficiency Impairs Activated T Cell Death and Enhances T Cell Survival

CRACR2A-Mediated TCR Signaling Promotes Local Effector Th1 and Th17 Responses

Computational Design of Potent D-Peptide Inhibitors of SARS-CoV-2

Calcium Signaling via Orai1 Is Essential for Induction of the Nuclear Orphan Receptor Pathway To Drive Th17 Differentiation