Jonathan Witton

Ferroptosis: past, present and future

The flavonoid quercetin ameliorates Alzheimer's disease pathology and protects cognitive and emotional function in aged triple transgenic Alzheimer's disease model mice

Microglial cell origin and phenotypes in health and disease

Ageing as a risk factor for neurodegenerative disease

The molecular machinery of regulated cell death

Developmental neuroplasticity and the origin of neurodegenerative diseases

Host microbiota constantly control maturation and function of microglia in the CNS

Oxytosis/Ferroptosis—(Re-) Emerging Roles for Oxidative Stress-Dependent Non-apoptotic Cell Death in Diseases of the Central Nervous System

Gene expression dysregulation domains are not a specific feature of Down syndrome

Down syndrome

Natural Polyphenols for Prevention and Treatment of Cancer

Therapeutic Potential of Quercetin: New Insights and Perspectives for Human Health

Receptors for Purines and Pyrimidines

Microglial M1/M2 polarization and metabolic states

Rodent models in Down syndrome research: impact and future opportunities

Apigenin induces apoptosis in human lung cancer H460 cells through caspase- and mitochondria-dependent pathways

Physiology of Microglia

Altered Synapse Stability in the Early Stages of Tauopathy

Increase in the density of resting microglia precedes neuritic plaque formation and microglial activation in a transgenic model of Alzheimer's disease

The functional neurophysiology of the amyloid precursor protein (APP) processing pathway

Hippocampal circuit dysfunction in the Tc1 mouse model of Down syndrome

Increased densities of resting and activated microglia in the dentate gyrus follow senile plaque formation in the CA1 subfield of the hippocampus in the triple transgenic model of Alzheimer's disease