J.F. Subra

Treml4, an Ig Superfamily Member, Mediates Presentation of Several Antigens to T Cells In Vivo, Including Protective Immunity to HER2 Protein

The use of a DNA biobank linked to electronic medical records to characterize pharmacogenomic predictors of tacrolimus dose requirement in kidney transplant recipients

Clinical Pharmacogenetics Implementation Consortium Guidelines for CYP2C19 Genotype and Clopidogrel Therapy: 2013 Update

Neutrophils at work

Neutrophil clearance: when the party is over, clean-up begins

Interplay between innate immunity and Alzheimer disease: APOE and TREM2 in the spotlight

Neutrophils in the activation and regulation of innate and adaptive immunity

PharmGKB summary

Alzheimer Disease: An Update on Pathobiology and Treatment Strategies

TFOS DEWS II Management and Therapy Report

Molecular mechanisms of necroptosis: an ordered cellular explosion

Cytochrome P450 enzymes in drug metabolism: Regulation of gene expression, enzyme activities, and impact of genetic variation

The acute respiratory distress syndrome

Molecular mechanisms of late apoptotic/necrotic cell clearance

Neutrophil recruitment and function in health and inflammation

Effect of a New FunctionalCYP3A4Polymorphism on Calcineurin Inhibitors‘ Dose Requirements and Trough Blood Levels in Stable Renal Transplant Patients

Practical Recommendations for Pharmacogenomics-based Prescription: 2010 ESF–UB Conference on Pharmacogenetics and Pharmacogenomics

CCR7 is involved in the migration of neutrophils to lymph nodes

Wound Healing: A Cellular Perspective

Using Genetic and Clinical Factors to Predict Tacrolimus Dose in Renal Transplant Recipients

A New Functional CYP3A4 Intron 6 Polymorphism Significantly Affects Tacrolimus Pharmacokinetics in Kidney Transplant Recipients

Optimization of Initial Tacrolimus Dose Using Pharmacogenetic Testing

Endogenous PTX3 translocates at the membrane of late apoptotic human neutrophils and is involved in their engulfment by macrophages

Clusterin facilitates apoptotic cell clearance and prevents apoptotic cell-induced autoimmune responses