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Works of J. Wayne Conlan being referenced
The major outer-membrane proteins of Chlamydia trachomatis serovars A and B: intra-serovar amino acid changes do not alter specificities of serovar- and C subspecies-reactive antibody-binding domains
1990
Cellular and humoral immunity are synergistic in protection against types A and B Francisella tularensis
2008
Susceptibility of three strains of conventional adult mice to intestinal colonization by an isolate of <i>Escherichia coli</i> O157:H7
1998
Functional genomics of Helicobacter pylori: identification of a β‐1,4 galactosyltransferase and generation of mutants with altered lipopolysaccharide
2000
Multidomain binding of transforming growth factor .alpha. to the epidermal growth factor receptor
1992
Neutrophils and tumour necrosis factor- are important for controlling early gastrointestinal stages of experimental murine listeriosis
1997
Chlamydia trachomatis major outer membrane protein epitopes expressed as fusions with LamB in an attenuated aroA strain of Salmonella typhimurium; their application as potential immunogens
1991
Structural analysis of Francisella tularensis lipopolysaccharide
2002
Early pathogenesis of Listeria monocytogenes infection in the mouse spleen
1996
Toll-like receptor 4 (TLR4) plays a relatively minor role in murine defense against primary intradermal infection with Francisella tularensis LVS
2004
Murine listeriosis as a model of antimicrobial defense
1997
In vivo Production of a Tissue-destructive Protease by Legionella pneumophila in the Lungs of Experimentally Infected Guinea-pigs
1988
Early pathogenesis of infection in the liver with the facultative intracellular bacteria Listeria monocytogenes, Francisella tularensis, and Salmonella typhimurium involves lysis of infected hepatocytes by leukocytes
1992
Neutrophil-mediated dissolution of infected host cells as a defense strategy against a facultative intracellular bacterium.
1991
Separation of Legionella pneumophila Proteases and Purification of a Protease Which Produces Lesions Like Those of Legionnaires" Disease in Guinea Pig Lung
1986
Neutrophils are essential for early anti-Listeria defense in the liver, but not in the spleen or peritoneal cavity, as revealed by a granulocyte-depleting monoclonal antibody.
1994
Immunity to <i>Listeria monocytogenes</i>
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Roles of Listeria monocytogenes virulence factors in survival: virulence factors distinct from listeriolysin are needed for the organism to survive an early neutrophil-mediated host defense mechanism
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Mouse Model of Oral Infection with Virulent Type A Francisella tularensis
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Pulmonary damage caused by a protease from Legionella pneumophila.
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Salmonella landau as a live vaccine against Escherichia coli O157:H7 investigated in a mouse model of intestinal colonization
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Neutrophils prevent extracellular colonization of the liver microvasculature by Salmonella typhimurium
1996
Susceptibility of three strains of conventional adult mice to intestinal colonization by an isolate of Escherichia coli O157:H7
1998
Monoclonal antibody NIMP-R10 directed against the CD11b chain of the type 3 complement receptor can substitute for monoclonal antibody 5C6 to exacerbate listeriosis by preventing the focusing of myelomonocytic cells at infectious foci in the liver
1992
Critical roles of neutrophils in host defense against experimental systemic infections of mice by Listeria monocytogenes, Salmonella typhimurium, and Yersinia enterocolitica
1997
Survival of virulentLegionella pneumophilain aerosols
1983
Leukocyte-mediated lysis of infected hepatocytes during listeriosis occurs in mice depleted of NK cells or CD4+ CD8+ Thy1.2+ T cells
1993
Neutrophils are critical for host defense against primary infection with the facultative intracellular bacterium Francisella tularensis in mice and participate in defense against reinfection
1994