J. Libby

Transforming growth factor–β3 is required for secondary palate fusion

Transforming growth factor beta (TGF-?) in inflammation: A cause and a cure

Transforming Growth Factor‐β

Growth factors and cutaneous wound repair

Role of Oxidative Modifications in Atherosclerosis

Effects of thioredoxin reductase-1 deletion on embryogenesis and transcriptome

Tyrosine Phosphorylation Regulates H2O2Production in Lung Fibroblasts Stimulated by Transforming Growth Factor β1

Targeted disruption of the mouse transforming growth factor-β1 gene results in multifocal inflammatory disease

The pathogenesis of atherosclerosis: a perspective for the 1990s

REGULATION OF IMMUNE RESPONSES BY TGF-β

TGF-β SIGNAL TRANSDUCTION

TGFβ2 knockout mice have multiple developmental defects that are non-overlapping with other TGFβ knockout phenotypes

Transforming growth factor-beta 1 induces alpha-smooth muscle actin expression in granulation tissue myofibroblasts and in quiescent and growing cultured fibroblasts

Reactive oxygen species in cell signaling

Apoptosis mediates the decrease in cellularity during the transition between granulation tissue and scar.

Some recent advances in the chemistry and biology of transforming growth factor-beta.

The high molecular weight receptor to transforming growth factor-beta contains glycosaminoglycan chains.

Transforming growth factor beta 1 null mutation in mice causes excessive inflammatory response and early death.

Regulation of Wound Healing by Growth Factors and Cytokines

LEF-1 is crucial for neutrophil granulocytopoiesis and its expression is severely reduced in congenital neutropenia

Tyrosine phosphorylation in cells treated with transforming growth factor‐β