Garima Singh

Topological control of cytokine receptor signaling induces differential effects in hematopoiesis

Acetyl-CoA Metabolism Supports Multistep Pancreatic Tumorigenesis

Bone Morphogenetic Protein (BMP) signaling in development and human diseases

A pupal transcriptomic screen identifies Ral as a target of store-operated calcium entry in Drosophila neurons

Identifying Functional Hotspot Residues for Biased Ligand Design in G-Protein-Coupled Receptors

Mutant p53 induces a hypoxia transcriptional program in gastric and esophageal adenocarcinoma

Store-Operated CRAC Channels Regulate Gene Expression and Proliferation in Neural Progenitor Cells

NFAT, immunity and cancer: a transcription factor comes of age

SCENIC: single-cell regulatory network inference and clustering

EMT Transition States during Tumor Progression and Metastasis

Molecular Dynamics Simulation for All

Acinar cell plasticity and development of pancreatic ductal adenocarcinoma

Population Heterogeneity in the Epithelial to Mesenchymal Transition Is Controlled by NFAT and Phosphorylated Sp1

From Krebs to clinic: glutamine metabolism to cancer therapy

NFATc2 is an intrinsic regulator of melanoma dedifferentiation

Reactive Oxygen Species: Involvement in T Cell Signaling and Metabolism

Role of Smads in TGFβ signaling

Acetyl-CoA promotes glioblastoma cell adhesion and migration through Ca²⁺–NFAT signaling

<p>Reactive Oxygen Species: Drivers of Physiological and Pathological Processes</p>

Mitochondrial TCA cycle metabolites control physiology and disease

Biased ligand quantification in drug discovery: from theory to high throughput screening to identify new biased μ opioid receptor agonists

Follicular Helper CD4 T Cells (T_FH)

Metabolic reprogramming and cancer progression

Simultaneously Targeting Tissue Transglutaminase and Kidney Type Glutaminase Sensitizes Cancer Cells to Acid Toxicity and Offers New Opportunities for Therapeutic Intervention

A Mechanism for the Upregulation of EGF Receptor Levels in Glioblastomas

Emerging structural insights into biased GPCR signaling

Sequential Activation of NFAT and c-Myc Transcription Factors Mediates the TGF-β Switch from a Suppressor to a Promoter of Cancer Cell Proliferation

NFAT-Induced Histone Acetylation Relay Switch Promotes c-Myc-Dependent Growth in Pancreatic Cancer Cells

NFATc1 Links EGFR Signaling to Induction of Sox9 Transcription and Acinar–Ductal Transdifferentiation in the Pancreas