Fu Han

Transition from inflammation to proliferation: a critical step during wound healing

Microvesicles from human adipose stem cells promote wound healing by optimizing cellular functions via AKT and ERK signaling pathways

Macrophages in immunoregulation and therapeutics

Methods Used to Evaluate Pain Behaviors in Rodents

Skin Wound Healing: An Update on the Current Knowledge and Concepts

The JAK/STAT signaling pathway: from bench to clinic

Reducing CXCR4-mediated nociceptor hyperexcitability reverses painful diabetic neuropathy

The Immune Functions of Keratinocytes in Skin Wound Healing

Functional characterization of a mouse model for central post-stroke pain

The Role of Voltage-Gated Sodium Channels in Pain Signaling

Artemisinin as an anticancer drug: Recent advances in target profiling and mechanisms of action

The Role of Notch Signaling in Macrophages during Inflammation and Infection: Implication in Rheumatoid Arthritis?

Neuropathic Pain: From Mechanisms to Treatment

Immunology of Acute and Chronic Wound Healing

SDF1–CXCR4 signaling contributes to persistent pain and hypersensitivity via regulating excitability of primary nociceptive neurons: involvement of ERK-dependent Nav1.8 up-regulation

Cell-free therapy based on adipose tissue stem cell-derived exosomes promotes wound healing via the PI3K/Akt signaling pathway

Dihydroartemisinin increases temozolomide efficacy in glioma cells by inducing autophagy

Post-stroke pain hypersensitivity induced by experimental thalamic hemorrhage in rats is region-specific and demonstrates limited efficacy of gabapentin

Acute downregulation of miR-155 at wound sites leads to a reduced fibrosis through attenuating inflammatory response

MicroRNA-146a protects against LPS-induced organ damage by inhibiting Notch1 in macrophage