Daniela Hoehn

Staging and grading of periodontitis: Framework and proposal of a new classification and case definition

The 2016 revision of the World Health Organization classification of lymphoid neoplasms

The promise and challenge of therapeutic genome editing

Follicular lymphoma-like B cells of uncertain significance (in situ follicular lymphoma) may infrequently progress, but precedes follicular lymphoma, is associated with other overt lymphomas and mimics follicular lymphoma in flow cytometric studies

SETBP1 mutations occur in 9% of MDS/MPN and in 4% of MPN cases and are strongly associated with atypical CML, monosomy 7, isochromosome i(17)(q10), ASXL1 and CBL mutations

Cancer stem cells revisited

Epigenetics in Cancer: A Hematological Perspective

Anchor extension: a structure-guided approach to design cyclic peptides targeting enzyme active sites

An International MDS/MPN Working Group's perspective and recommendations on molecular pathogenesis, diagnosis and clinical characterization of myelodysplastic/myeloproliferative neoplasms

A new classification scheme for periodontal and peri‐implant diseases and conditions – Introduction and key changes from the 1999 classification

Non–plaque‐induced gingival diseases

Non–plaque‐induced gingival diseases

Multiplexed genetic engineering of human hematopoietic stem and progenitor cells using CRISPR/Cas9 and AAV6

HDACs and HDAC Inhibitors in Cancer Development and Therapy

CRISPR-Cas guides the future of genetic engineering

The 2016 revision to the World Health Organization classification of myeloid neoplasms and acute leukemia

Dual Targeting of Protein Degradation Pathways with the Selective HDAC6 Inhibitor ACY-1215 and Bortezomib Is Synergistic in Lymphoma

Classical Hodgkin Lymphoma Arising in the Setting of Iatrogenic Immunodeficiency

Lymphoid follicle colonization by Bcl-2bright+CD10+ B-cells (“follicular lymphoma in situ”) at nodal and extranodal sites can be a manifestation of follicular homing of lymphoma

Myeloid neoplasms with isolated isochromosome 17q represent a clinicopathologic entity associated with myelodysplastic/myeloproliferative features, a high risk of leukemic transformation, and wild‐type TP53

Cohesin loss alters adult hematopoietic stem cell homeostasis, leading to myeloproliferative neoplasms