Bernadette Chen

Activation of PAD4 in NET formation

A Critical Reappraisal of Neutrophil Extracellular Traps and NETosis Mimics Based on Differential Requirements for Protein Citrullination

Neutrophils at work

Infection-induced NETosis is a dynamic process involving neutrophil multitasking in vivo

In vitro induction of NETosis: Comprehensive live imaging comparison and systematic review

The molecular machinery of regulated cell death

Dynamic transcriptomic m6A decoration: writers, erasers, readers and functions in RNA metabolism

Immunometabolism within the tuberculosis granuloma: amino acids, hypoxia, and cellular respiration

Neutrophil extracellular traps in immunity and disease

Reconstructing sickle cell disease: A data‐based analysis of the “hyperhemolysis paradigm” for pulmonary hypertension from the perspective of evidence‐based medicine

Sickle-cell disease

Molecular mechanisms regulating NETosis in infection and disease

Neutrophil recruitment and function in health and inflammation

Hypoxia‐dependent mRNA expression pattern of splicing factor YT521 and its impact on oncological important target gene expression

Macrophage Polarization

Reactive Oxygen Species and Antioxidant Defense in Plants under Abiotic Stress: Revisiting the Crucial Role of a Universal Defense Regulator

Synthetic Siglec-9 Agonists Inhibit Neutrophil Activation Associated with COVID-19

Klebsiella pneumoniae: Going on the Offense with a Strong Defense

The role of inflammation in hypoxic pulmonary hypertension: from cellular mechanisms to clinical phenotypes

Controlled Oxygen Release from Pyridone Endoperoxides Promotes Cell Survival under Anoxic Conditions

Adventitial Fibroblasts Induce a Distinct Proinflammatory/Profibrotic Macrophage Phenotype in Pulmonary Hypertension

Early Macrophage Recruitment and Alternative Activation Are Critical for the Later Development of Hypoxia-Induced Pulmonary Hypertension

Hypoxia-Inducible Factor 1α Is a Critical Downstream Mediator for Hypoxia-Induced Mitogenic Factor (FIZZ1/RELMα)–Induced Pulmonary Hypertension

Immunological mechanisms contributing to the double burden of diabetes and intracellular bacterial infections

Reactive oxygen species generating systems meeting challenges of photodynamic cancer therapy

Antioxidant System and Biomolecules Alteration in Pisum sativum under Heavy Metal Stress and Possible Alleviation by 5-Aminolevulinic Acid

Mice deficient in Mkp-1 develop more severe pulmonary hypertension and greater lung protein levels of arginase in response to chronic hypoxia

Glutathione reductase facilitates host defense by sustaining phagocytic oxidative burst and promoting the development of neutrophil extracellular traps (172.3)

Arginase II is a target of miR-17-5p and regulates miR-17-5p expression in human pulmonary artery smooth muscle cells

Glutathione Reductase Facilitates Host Defense by Sustaining Phagocytic Oxidative Burst and Promoting the Development of Neutrophil Extracellular Traps

Hypoxia-induced proliferation of human pulmonary microvascular endothelial cells depends on epidermal growth factor receptor tyrosine kinase activation

Hypoxia promotes human pulmonary artery smooth muscle cell proliferation through induction of arginase