Andreas Barratt‐Due

Complement, a target for therapy in inflammatory and degenerative diseases

Complement as a diagnostic tool in immunopathology

Toxic Mechanisms of Five Heavy Metals: Mercury, Lead, Chromium, Cadmium, and Arsenic

Chronic low level arsenic exposure evokes inflammatory responses and DNA damage

Be on Target: Strategies of Targeting Alternative and Lectin Pathway Components in Complement-Mediated Diseases

Complement: a key system for immune surveillance and homeostasis

Complement associated microvascular injury and thrombosis in the pathogenesis of severe COVID-19 infection: A report of five cases

Inflammation and tumor progression: signaling pathways and targeted intervention

Immunohaemostasis: a new view on haemostasis during sepsis

The immunopathology of sepsis and potential therapeutic targets

Thrombosis as an intravascular effector of innate immunity

Pathogenic, immunologic, and clinical aspects of sepsis – update 2016

Complement inhibition decreases the procoagulant response and confers organ protection in a baboon model of Escherichia coli sepsis

Complement therapeutics in inflammatory diseases: promising drug candidates for C3‐targeted intervention

Molecular Docking: Shifting Paradigms in Drug Discovery

A meta-analysis of randomized controlled trials

Targeting the complement system in bacterial meningitis

COVID-19 and its implications for thrombosis and anticoagulation

Repurposing mosloflavone/5,6,7-trimethoxyflavone-resveratrol hybrids: Discovery of novel p38-α MAPK inhibitors as potent interceptors of macrophage-dependent production of proinflammatory mediators

The oral microbiota: dynamic communities and host interactions

Complement in clinical medicine: Clinical trials, case reports and therapy monitoring

Selective inhibition of TNF-α or IL-1β does not affect E. coli-induced inflammation in human whole blood

Combined inhibition of complement and CD14 improved outcome in porcine polymicrobial sepsis

The Role of Bradykinin and the Effect of the Bradykinin Receptor Antagonist Icatibant in Porcine Sepsis

Double Blockade of CD14 and Complement C5 Abolishes the Cytokine Storm and Improves Morbidity and Survival in Polymicrobial Sepsis in Mice

Post challenge inhibition of C3 and CD14 attenuates Escherichia coli-induced inflammation in human whole blood

CD14 inhibition efficiently attenuates early inflammatory and hemostatic responses in Escherichia coli sepsis in pigs